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February 25, 2016 by admin 2 Comments

Testosterone Boosting Smoothie Recipe that Works! 5 (1)

Testosterone Boosting Smoothie Recipe that works!

Testosterone Boosting Smoothie recipes can be found all over the place. For the most part, instructions are to mix a couple of fruits and vegetables together to make a healthy meal replacement drink but, the ingredients involved, are they really focused on boosting our testosterone levels? Are there Testosterone boosting smoothie recipes out there that really ignite our natural production of testosterone? For a testosterone boosting smoothie to really be talked about and recommended, whoever is ingesting it has to be able to feel a significant difference in their body. They have to be able to easily detect that the ingredients fulfilled the promise of boosting their main male hormone. It’s an undeniable surge when testosterone levels are flowing appropriately.

Well, we have just that smoothie for you, and a list of the ingredients and why they do what they do to fulfill your testosterone boosting needs.

This testosterone-boosting smoothie can help boost testosterone levels by almost 20%. It’s not the most delicious smoothie in the world but, you may be looking at it all wrong. Just remember, nothing tastes more delicious than feeling strong and sexy. That is what testosterone does for you…. Try it out for a week! It will change your life.

Testosterone Boosting SmoothieTestosterone Boosting Smoothie

Testosterone Boosting Smoothie

We will be incorporating a series of testosterone boosting smoothie recipes to help our interested readers find a natural way of boosting their testosterone. Sure, nothing is more effective at raising testosterone levels that an injectable testosterone therapy program but, why jump right into therapy before first trying to help your body to naturally produce the required.

The thing that most people don’t understand about being on testosterone therapy is that, once you start, you pretty much have to keep going in order to continue feeling as wonderful as your therapy made you feel. You will never naturally reproduce the attainable levels of an injectable program so, before finally deciding on a more “involved”, therapeutic protocol for increasing testosterone levels, try by all measures to incite your own natural production. Once you find that you cannot raise your testosterone levels naturally beyond 550 ng/dl, for your own well-being and the future of your well-being, then it’s time to start looking at more powerful alternatives, such as injectable testosterone therapy.

Testosterone Therapy Information

February 10, 2016 by admin 0 Comments

Is Testosterone Therapy Preventive Medicine? 5 (2)

Is Testosterone Therapy Preventive Medicine?

testosterone therapy preventive medicine

Is Testosterone therapy preventive medicine? This is a very fair question. We hear of so many pro’s and cons but, is there an actual medical need for testosterone. Is Testosterone therapy preventive medicine or, where does it rank in terms of the necessity for the male body?

It could be said that testosterone is what makes men, men. It gives them:

  • their characteristic deep voices
  • larger muscles
  • facial and body hair, distinguishing them from women.

It also:

  • stimulates the growth of the genitals at puberty
  • plays a role in sperm production
  • fuels libido
  • contributes to normal erections
  • fosters the production of red blood cells
  • boosts mood
  • aids cognition

Over time, the testicular “machinery” that makes testosterone gradually becomes less effective, and testosterone levels start to fall, by about 1% a year, beginning in the 40s. As men get into their 50s, 60s, and beyond, they may start to have signs and symptoms of low testosterone such as:

  • lower sex drive and sense of vitality
  • erectile dysfunction
  • decreased energy
  • reduced muscle mass and bone density
  • and even anemia, to name a few.

Taken together, these signs and symptoms are often called hypogonadism (“hypo” meaning low functioning and “gonadism” referring to the testicles). Researchers estimate that the condition affects anywhere from two to six million men in the United States. Yet it is an underdiagnosed problem, with only about 5% of those affected receiving treatment.

Studies have shown that testosterone-replacement therapy may offer a wide range of benefits for men with hypogonadism, including improved libido, mood, cognition, muscle mass, bone density, and red blood cell production.

Taking into account some of the scientific facts delineated above, its simple to see how maintaining healthy testosterone levels can help in preventing accelerated aging. If maintaining healthy testosterone levels is reflected in a patient maintaining their vigor, strength, sexual interest and functionality and positive attitude, then it should be deemed preventive medicine. We think we can say “yes”. Testosterone therapy is preventive medicine.

Consider the fact that Testosterone is an incomparable antioxidant to the heart and its proven to maintain and aid in brain health. Not only do these points make testosterone out to be preventive medicine, it proves it an invaluable aspect of a male’s anti-aging regimen. No matter how well off all your other hormones are, as a male, if your testosterone is low, you are just simply not your normal self… and you can feel it. Call us at your earliest convenience so we can help you determine your testosterone levels. We even have

You can also reach us by filling out our Medical History Form. Upon completion, you can expect a contact within 24 business hours from one of our expert Wellness Advisors. Take advantage, testosterone therapy preventive medicine is our area of expertise. Let us share more life-transforming fact as they would affect you specifically. Contact us now.

 

testosterone-injections

 

Testosterone Injections is the most common treatment for men going through andropause. This therapy may provide help and relief from the symptoms and help improve the quality of life in many cases, also lifestyle changes such as increased exercise, stress reduction, and proper nutrition also help.

Testosterone therapy is available in different forms, ask your doctor he will help determine which treatment is best for you.

TESTOSTERONE INJECTIONS: This treatment involves doses of bioidentical (Testosterone Cypionate, Testosterone Enanthate, and Testosterone Propionate).

TESTOSTERONE PATCHES: People who wear a piece containing testosterone receive the hormone through the skin. The patches allow a slow, steady release of testosterone into the bloodstream.

TESTOSTERONE GEL: This treatment is also applied directly to the skin, usually on the arms. Because the gel may transfer to other individuals through skin contact, a person must take care to wash the gel from the hands after each application.

TESTOSTERONE CAPSULES: This is yet another option for testosterone replacement. Men with liver disease, poor liver function, severe heart or kidney disease, or too much calcium in their blood should avoid testosterone capsules.

Follow-up visits with your doctor will be necessary after the initial treatment begins. At follow-up visits, your doctor will check your response to the treatment and make adjustments, if necessary.

February 4, 2016 by admin 0 Comments

Depo Testosterone Description and Information 5 (1)

Depo Testosterone

Depo Testosterone Injection, for intramuscular injection, contains testosterone cypionate which is the oil-soluble 17 (beta)- cyclopentyl propionate ester of the androgenic hormone testosterone.

Testosterone cypionate is a white or creamy white crystalline powder, odorless or nearly so and stable in air. It is insoluble in water, freely soluble in alcohol, chloroform, dioxane, ether, and soluble in vegetable oils.

The chemical name for testosterone cypionate is androst-4-en-3-one, 17-(3-cyclopentyl-1-oxopropoxy)-, (17β)-. Its molecular formula is C27H40O3, and the molecular weight 412.61.

The structural formula is represented below:


Depo-Testosterone Injection is available in two strengths, 100 mg/mL, and 200 mg/mL testosterone cypionate.

Each mL of the 100 mg/mL solution contains:
Testosterone cypionate……………………………………………………………………….. 100 mg
Benzyl benzoate …………………………………………………………………………………. 0.1 mL
Cottonseed oil …………………………………………………………………………………… 736 mg
Benzyl alcohol (as preservative) …………………………………………………………… 9.45 mg

Each mL of the 200 mg/mL solution contains:
Testosterone cypionate ………………………………………………………………………. 200 mg
Benzyl benzoate …………………………………………………………………………………. 0.2 mL
Cottonseed oil …………………………………………………………………………………… 560 mg
Benzyl alcohol (as preservative) …………………………………………………………… 9.45 mg

Depo Testosterone – Clinical Pharmacology
Endogenous androgens are responsible for normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include growth and maturation of the prostate, seminal vesicles, penis, and scrotum; development of male hair distribution, such as beard, pubic, chest, and axillary hair; laryngeal enlargement, vocal cord thickening, and alterations in body musculature and fat distribution. Drugs in this class also cause retention of nitrogen, sodium, potassium, and phosphorous, and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism. Nitrogen balance is improved only when there is sufficient intake of calories and protein.

Androgens are responsible for the growth spurt of adolescence and for eventual termination of linear growth, brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates but may cause a disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of the growth process. Androgens have been reported to stimulate the production of red blood cells by enhancing production of erythropoietic stimulation factor.

Slideshow: Welcome to Parenthood! 10 Things To Prepare Yourself For Welcome to Parenthood! 10 Things To Prepare Yourself For:
During exogenous administration of androgens, endogenous testosterone release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH). At large doses of exogenous androgens, spermatogenesis may also be suppressed through feedback inhibition of pituitary follicle-stimulating hormone (FSH).

There is a lack of substantial evidence that androgens are effective in fractures, surgery, convalescence, and functional uterine bleeding.

Pharmacokinetics
Testosterone esters are less polar than free testosterone. Testosterone esters in oil injected intramuscularly are absorbed slowly from the lipid phase; thus, testosterone cypionate can be given at intervals of two to four weeks.

Testosterone in plasma is 98 percent bound to a specific testosterone-estradiol binding globulin, and about 2 percent is free. Generally, the amount of this sex-hormone binding globulin in the plasma will determine the distribution of testosterone between free and bound forms, and the free testosterone concentration will determine its half-life.

About 90 percent of a dose of testosterone is excreted in the urine as glucuronic and sulfuric acid conjugates of testosterone and its metabolites; about 6 percent of a dose is excreted in the feces, mostly in the unconjugated form. Inactivation of testosterone occurs primarily in the liver. Testosterone is metabolized to various 17-keto steroids through two different pathways.

The half-life of testosterone cypionate, when injected intramuscularly, is approximately eight days.

In many tissues, the activity of testosterone therapy appears to depend on reduction to dihydrotestosterone, which binds to cytosol receptor proteins. The steroid-receptor complex is transported to the nucleus where it initiates transcription events and cellular changes related to androgen action.

Indications and Usage for Depo Testosterone

Depo Testosterone Injection is indicated for replacement therapy in the male in conditions associated with symptoms of deficiency or absence of endogenous testosterone.

1. Primary hypogonadism (congenital or acquired)-testicular failure due to cryptorchidism, bilateral torsion, orchitis, vanishing testis syndrome; or orchidectomy.

2. Hypogonadotropic hypogonadism (congenital or acquired)- gonadotropin or LHRH deficiency, or pituitary-hypothalamic injury from tumors, trauma, or radiation.

Safety and efficacy of Depo Testosterone (testosterone cypionate) in men with “age-related hypogonadism” (also referred to as “late-onset hypogonadism”) have not been established.

Contraindications

  1. Known hypersensitivity to the drug
  2. Males with carcinoma of the breast
  3. Males with known or suspected carcinoma of the prostate gland
  4. Women who are or who may become pregnant
  5. Patients with serious cardiac, hepatic or renal disease

Warnings

Hypercalcemia may occur in immobilized patients. If this occurs, the drug should be discontinued.

Prolonged use of high doses of androgens (principally the 17-α alkyl-androgens) has been associated with the development of hepatic adenomas, hepatocellular carcinoma, and peliosis hepatis —all potentially life-threatening complications.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

There have been postmarketing reports of venous thromboembolic events, including deep vein thrombosis (DVT) and pulmonary embolism (PE), in patients using testosterone products, such as testosterone cypionate. Evaluate patients who report symptoms of pain, edema, warmth, and erythema in the lower extremity for DVT and those who present with acute shortness of breath for PE. If a venous thromboembolic event is suspected, discontinue treatment with testosterone cypionate and initiate appropriate workup and management.

Long-term clinical safety trials have not been conducted to assess the cardiovascular outcomes of testosterone replacement therapy in men. To date, epidemiologic studies and randomized controlled trials have been inconclusive for determining the risk of major adverse cardiovascular events (MACE), such as non-fatal myocardial infarction, non-fatal stroke, and cardiovascular death, with the use of testosterone compared to non-use. Some studies, but not all, have reported an increased risk of MACE in association with the use of testosterone replacement therapy in men. Patients should be informed of this possible risk when deciding whether to use or to continue to use Depo Testosterone (testosterone cypionate).

Edema, with or without congestive heart failure, may be a serious complication in patients with pre-existing cardiac, renal or hepatic disease.

Gynecomastia may develop and occasionally persists in patients being treated for hypogonadism.

The preservative benzyl alcohol has been associated with serious adverse events, including the “gasping syndrome”, and death in pediatric patients. Although normal therapeutic doses of this product ordinarily deliver amounts of benzyl alcohol that are substantially lower than those reported in association with the “gasping syndrome”, the minimum amount of benzyl alcohol at which toxicity may occur is not known. The risk of benzyl alcohol toxicity depends on the quantity administered and the hepatic capacity to detoxify the chemical. Premature and low-birth weight infants may be more likely to develop toxicity.

Androgen therapy should be used cautiously in healthy males with delayed puberty. The effect on bone maturation should be monitored by assessing the bone age of the wrist and hand every 6 months. In children, androgen treatment may accelerate bone maturation without producing the compensatory gain in linear growth. This adverse effect may result in compromised adult stature. The younger the child the greater the risk of compromising final mature height.

This drug has not been shown to be safe and effective for the enhancement of athletic performance. Because of the potential risk of serious adverse health effects, this drug should not be used for such purpose.

Precautions

General

Patients with benign prostatic hypertrophy may develop acute urethral obstruction. Priapism or excessive sexual stimulation may develop. Oligospermia may occur after prolonged administration or excessive dosage. If any of these effects appear, the androgen should be stopped and if restarted, a lower dosage should be utilized.

Testosterone cypionate should not be used interchangeably with testosterone propionate because of differences in duration of action.

Testosterone cypionate is not for intravenous use.

Information for patients

Patients should be instructed to report any of the following: nausea, vomiting, changes in skin color, ankle swelling, too frequent or persistent erections of the penis.

Laboratory tests

Hemoglobin and hematocrit levels (to detect polycythemia) should be checked periodically in patients receiving long-term androgen administration.

Serum cholesterol may increase during androgen therapy.

Drug interactions

Androgens may increase sensitivity to oral anticoagulants. The dosage of the anticoagulant may require the reduction in order to maintain satisfactory therapeutic hypoprothrombinemia.

Concurrent administration of oxyphenbutazone and androgens may result in elevated serum levels of oxyphenbutazone.

In diabetic patients, the metabolic effects of androgens may decrease blood glucose and, therefore, insulin requirements.

Drug/Laboratory test Interferences

Androgens may decrease levels of thyroxine-binding globulin, resulting in decreased total T4 serum levels and increased resin uptake of T3 and T4. Free thyroid hormone levels remain unchanged, however, and there is no clinical evidence of thyroid dysfunction.

Carcinogenesis

Animal data

Testosterone has been tested by subcutaneous injection and implantation in mice and rats. The implant induced cervical-uterine tumors in mice, which metastasized in some cases. There is suggestive evidence that injection of testosterone into some strains of female mice increases their susceptibility to hepatoma. Testosterone is also known to increase the number of tumors and decrease the degree of differentiation of chemically induced carcinomas of the liver in rats.

Human data

There are rare reports of hepatocellular carcinoma in patients receiving long-term therapy with androgens in high doses. Withdrawal of the drugs did not lead to regression of the tumors in all cases.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

Pregnancy

Teratogenic Effects

Pregnancy Category X. (See CONTRAINDICATIONS.)

Benzyl alcohol can cross the placenta. See WARNINGS.

Nursing Mothers

Depo Testosterone is not recommended for use in nursing mothers.

Pediatric use

Safety and effectiveness in pediatric patients below the age of 12 years have not been established.

Adverse Reactions

The following adverse reactions in the male have occurred with some androgens:

Endocrine and urogenital: Gynecomastia and excessive frequency and duration of penile erections. Oligospermia may occur at high dosages.

Skin and appendages: Hirsutism, male pattern of baldness, seborrhea, and acne.

Cardiovascular Disorders: myocardial infarction, stroke.

Fluid and electrolyte disturbances: Retention of sodium, chloride, water, potassium, calcium, and inorganic phosphates.

Gastrointestinal: Nausea, cholestatic jaundice, alterations in liver function tests, rarely hepatocellular neoplasms and peliosis hepatis (see WARNINGS).

Hematologic: Suppression of clotting factors II, V, VII, and X, bleeding in patients on concomitant anticoagulant therapy, and polycythemia.

Nervous system: Increased or decreased libido, headache, anxiety, depression, and generalized paresthesia.

Allergic: Hypersensitivity, including skin manifestations and anaphylactoid reactions.

Vascular disorders: Venous thromboembolism.

Miscellaneous: Inflammation and pain at the site of intramuscular injection.

Drug Abuse and Dependence

Controlled Substance Class

Testosterone is a controlled substance under the Anabolic Steroids Control Act, and Depo Testosterone Injection has been assigned to Schedule III.

Overdosage

There have been no reports of acute overdosage with the androgens.

Depo Testosterone Dosage and Administration

Prior to initiating Depo Testosterone (testosterone cypionate), confirm the diagnosis of hypogonadism by ensuring that serum testosterone concentrations have been measured in the morning on at least two separate days and that these serum testosterone concentrations are below the normal range.

Depo Testosterone Injection is for intramuscular use only.

It should not be given intravenously. Intramuscular injections should be given deep in the gluteal muscle.

The suggested dosage for Depo Testosterone Injection varies depending on the age, sex, and diagnosis of the individual patient. Dosage is adjusted according to the patient’s response and the appearance of adverse reactions.

Various dosage regimens have been used to induce pubertal changes in hypogonadal males; some experts have advocated lower dosages initially, gradually increasing the dose as puberty progresses, with or without a decrease to maintenance levels. Other experts emphasize that higher dosages are needed to induce pubertal changes and lower dosages can be used for maintenance after puberty. The chronological and skeletal ages must be taken into consideration, both in determining the initial dose and in adjusting the dose.

For replacement in the hypogonadal male, 50400 mg should be administered every two to four weeks.

Parenteral drug products should be inspected visually for particulate matter and discoloration prior to administration, whenever solution and container permit. Warming and shaking the vial should redissolve any crystals that may have formed during storage at temperatures lower than recommended.

How is Depo Testosterone Supplied

Depo Testosterone Injections is available as follows:

100 mg/mL
10 mL vials                    NDC 0009-0347-02

200 mg/mL
1 mL vials                      NDC 0009-0417-01
10 mL vials                    NDC 0009-0417-02

Vials should be stored at controlled room temperature 20° to 25°C (68° to 77°F) [see USP]. Protect from light.

February 4, 2016 by admin 0 Comments

Testosterone Cypionate Description 5 (1)

Testosterone Cypionate description injections

Testosterone Cypionate Description injection for intramuscular injection contains Testosterone Cypionate which is the oil-soluble 17 (beta)- cyclopentyl propionate ester of the androgenic hormone testosterone. Testosterone Cypionate is a white or creamy white crystalline powder, odorless or nearly so and stable in air. It is insoluble in water, freely soluble in alcohol, chloroform, dioxane, ether, and soluble in vegetable oils. The chemical name for Testosterone Cypionate is androst-4-en-3-one,17-(3-cyclopentyl-1-oxopropoxy)-, (17β)-. Its molecular formula is C27H40O3, and the molecular weight 412.61.

The structural formula is represented below:

Testosterone Cypionate injection, USP is available in two strengths, 100 mg/mL and 200 mg/mL Testosterone Cypionate, USP.

Each mL of the 100 mg/mL solution contains:

Testosterone Cypionate……………………………………………………………………. 100 mg
Benzyl benzoate ……………………………………………………………………………… 0.1 mL
Cottonseed oil ………………………………………………………………………………… 736 mg
Benzyl alcohol (as preservative)………………………………………………………… 9.45 mg

Each mL of the 200 mg/mL solution contains:

Testosterone Cypionate……………………………………………………………………. 200 mg
Benzyl benzoate………………………………………………………………………………. 0.2 mL
Cottonseed oil………………………………………………………………………………… 560 mg

Benzyl alcohol (as preservative)………………………………………………………… 9.45 mg

Testosterone Cypionate – Clinical Pharmacology

Endogenous androgens are responsible for normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include growth and maturation of the prostate, seminal vesicles, penis, and scrotum; development of male hair distribution, such as beard, pubic, chest, and axillary hair; laryngeal enlargement, vocal cord thickening, and alterations in body musculature and fat distribution. Drugs in this class also cause retention of nitrogen, sodium, potassium, and phosphorous, and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism. Nitrogen balance is improved only when there is sufficient intake of calories and protein.

Androgens are responsible for the growth spurt of adolescence and for eventual termination of linear growth, brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates but may cause a disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of the growth process. Androgens have been reported to stimulate the production of red blood cells by enhancing production of erythropoietic stimulation factor.

During exogenous administration of androgens, endogenous testosterone release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH). At large doses of exogenous androgens, spermatogenesis may also be suppressed through feedback inhibition of pituitary follicle stimulating hormone (FSH).

There is a lack of substantial evidence that androgens are effective in fractures, surgery, convalescence, and functional uterine bleeding.

Pharmacokinetics

Testosterone esters are less polar than free testosterone. Testosterone esters in oil injected intramuscularly are absorbed slowly from the lipid phase; thus, Testosterone Cypionate can be given at intervals of two to four weeks.

Testosterone in plasma is 98 percent bound to a specific testosterone-estradiol binding globulin, and about 2 percent is free. Generally, the amount of this sex-hormone binding globulin in the plasma will determine the distribution of testosterone between free and bound forms, and the free testosterone concentration will determine its half-life.

About 90 percent of a dose of testosterone injections is excreted in the urine as glucuronic and sulfuric acid conjugates of testosterone and its metabolites; about 6 percent of a dose is excreted in the feces, mostly in the unconjugated form. Inactivation of testosterone occurs primarily in the liver. Testosterone therapy is metabolized to various 17-keto steroids through two different pathways.

The half-life of Testosterone Cypionate, when injected intramuscularly, is approximately eight days.

In many tissues, the activity of testosterone therapy appears to depend on reduction to dihydrotestosterone, which binds to cytosol receptor proteins. The steroid-receptor complex is transported to the nucleus where it initiates transcription events and cellular changes related to androgen action.

Indications and Usage for Testosterone Cypionate description

Testosterone Cypionate description injection is indicated for replacement therapy in the male in conditions associated with symptoms of deficiency or absence of endogenous testosterone.

  • Primary hypogonadism (congenital or acquired)-testicular failure due to cryptorchidism, bilateral torsion, orchitis, vanishing testis syndrome; or orchidectomy.
  • Hypogonadotropic hypogonadism (congenital or acquired): gonadotropin or LHRH deficiency, or pituitary-hypothalamic injury from tumors, trauma, or radiation.

Safety and efficacy of Testosterone Cypionate description in men with “age-related hypogonadism” (also referred to as “late-onset hypogonadism”) have not been established.

Contraindications

  1. Known hypersensitivity to the drug
  2. Males with carcinoma of the breast
  3. Males with known or suspected carcinoma of the prostate gland
  4. Women who are or who may become pregnant
  5. Patients with serious cardiac, hepatic or renal disease

Warnings

Hypercalcemia may occur in immobilized patients. If this occurs, the drug should be discontinued.

Prolonged use of high doses of androgens (principally the 17-α alkyl-androgens) has been associated with the development of hepatic adenomas, hepatocellular carcinoma, and peliosis hepatis —all potentially life-threatening complications.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

There have been postmarketing reports of venous thromboembolic events, including deep vein thrombosis (DVT) and pulmonary embolism (PE), in patients using testosterone products, such as Testosterone Cypionate description. Evaluate patients who report symptoms of pain, edema, warmth, and erythema in the lower extremity for DVT and those who present with acute shortness of breath for PE. If a venous thromboembolic event is suspected, discontinue treatment with Testosterone Cypionate description and initiate appropriate workup and management.

Long term clinical safety trials have not been conducted to assess the cardiovascular outcomes of testosterone replacement therapy in men. To date, epidemiologic studies and randomized controlled trials have been inconclusive for determining the risk of major adverse cardiovascular events (MACE), such as non-fatal myocardial infarction, non-fatal stroke, and cardiovascular death, with the use of testosterone compared to non-use. Some studies, but not all, have reported an increased risk of MACE in association with the use of testosterone replacement therapy in men. Patients should be informed of this possible risk when deciding whether to use or to continue to use Testosterone Cypionate description.

Edema, with or without congestive heart failure, may be a serious complication in patients with preexisting cardiac, renal or hepatic disease.

Gynecomastia may develop and occasionally persists in patients being treated for hypogonadism.

The preservative benzyl alcohol has been associated with serious adverse events, including the “gasping syndrome”, and death in pediatric patients. Although normal therapeutic doses of this product ordinarily deliver amounts of benzyl alcohol that are substantially lower than those reported in association with the “gasping syndrome”, the minimum amount of benzyl alcohol at which toxicity may occur is not known. The risk of benzyl alcohol toxicity depends on the quantity administered and the hepatic capacity to detoxify the chemical. Premature and low-birth weight infants may be more likely to develop toxicity.

Androgen therapy should be used cautiously in healthy males with delayed puberty. The effect on bone maturation should be monitored by assessing the bone age of the wrist and hand every 6 months. In children, androgen treatment may accelerate bone maturation without producing a compensatory gain in linear growth. This adverse effect may result in compromised adult stature. The younger the child the greater the risk of compromising final mature height.

This drug has not been shown to be safe and effective for the enhancement of athletic performance. Because of the potential risk of serious adverse health effects, this drug should not be used for such purpose.

Precautions

General

Patients with benign prostatic hypertrophy may develop acute urethral obstruction. Priapism or excessive sexual stimulation may develop. Oligospermia may occur after prolonged administration or excessive dosage. If any of these effects appear, the androgen should be stopped and if restarted, a lower dosage should be utilized.

Testosterone Cypionate description should not be used interchangeably with testosterone propionate description because of differences in duration of action.

Testosterone Cypionate description is not for intravenous use.

Information for Patients

Patients should be instructed to report any of the following: nausea, vomiting, changes in skin color, ankle swelling, too frequent or persistent erections of the penis.

Laboratory Tests

Hemoglobin and hematocrit levels (to detect polycythemia) should be checked periodically in patients receiving long-term androgen administration.

Serum cholesterol may increase during androgen therapy.

Drug Interactions

Androgens may increase sensitivity to oral anticoagulants. The dosage of the anticoagulant may require a reduction in order to maintain satisfactory therapeutic hypoprothrombinemia.

Concurrent administration of oxyphenbutazone and androgens may result in elevated serum levels of oxyphenbutazone.

In diabetic patients, the metabolic effects of androgens may decrease blood glucose and, therefore, insulin requirements.

Drug/Laboratory Test Interferences

Androgens may decrease levels of thyroxine-binding globulin, resulting in decreased total T4 serum levels and increased resin uptake of T3 and T4. Free thyroid hormone levels remain unchanged, however, and there is no clinical evidence of thyroid dysfunction.

Carcinogenesis

Animal data

Testosterone has been tested by subcutaneous injection and implantation in mice and rats. The implant induced cervical-uterine tumors in mice, which metastasized in some cases. There is suggestive evidence that injection of testosterone into some strains of female mice increases their susceptibility to hepatoma. Testosterone is also known to increase the number of tumors and decrease the degree of differentiation of chemically induced carcinomas of the liver in rats.

Human data

There are rare reports of hepatocellular carcinoma in patients receiving long-term therapy with androgens in high doses. Withdrawal of the drugs did not lead to regression of the tumors in all cases.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

Pregnancy

Teratogenic Effects

Pregnancy Category X. (See CONTRAINDICATIONS)

Benzyl alcohol can cross the placenta. See WARNINGS.

Nursing Mothers

Testosterone Cypionate description is not recommended for use in nursing mothers.

Pediatric Use

Safety and effectiveness in pediatric patients below the age of 12 years have not been established.

Adverse Reactions

The following adverse reactions in the male have occurred with some androgens:

Endocrine and urogenital: Gynecomastia and excessive frequency and duration of penile erections. Oligospermia may occur at high dosages.

Skin and Appendages: Hirsutism, male pattern of baldness, seborrhea, and acne.

Cardiovascular Disorders – myocardial infarction, stroke

Fluid and electrolyte disturbances: Retention of sodium, chloride, water, potassium, calcium, and inorganic phosphates.

Gastrointestinal: Nausea, cholestatic jaundice, alterations in liver function tests, rarely hepatocellular neoplasms and peliosis hepatis (see WARNINGS).

Hematologic: Suppression of clotting factors II, V, VII, and X, bleeding in patients on concomitant anticoagulant therapy, and polycythemia.

Nervous system: Increased or decreased libido, headache, anxiety, depression, and generalized paresthesia.

Allergic: Hypersensitivity, including skin manifestations and anaphylactoid reactions.

Vascular Disorders: venous thromboembolism

Miscellaneous: Inflammation and pain at the site of intramuscular testosterone injection.

Testosterone Therapy Information

January 27, 2016 by Joseph Fermin 0 Comments

Where Does hGH Come From? 5 (2)

What is Human Growth Hormone?
Where Does hGH Come From?

What is human Growth hormone? Well, let’s start with the basics: Growth Hormone is the master hormone released by the pituitary gland. This peptide hormone is made up of 191 amino acids that form a long, single-chain polypeptide. Growth hormone is synthesized in the somatotropic cells, which, again, are found in the anterior pituitary gland. These cells are also responsible for storing and releasing the hormone. Growth hormone is a peptide hormone that stimulates growth, development, and regeneration. The pituitary gland is the master gland of the endocrine system. It controls all the glands of the endocrine system. Your hormones keep your entire body and all its systems functioning. Hormones impact your weight, hair, and nails, as well as sex drive and attitude. It’s essential to know your hormone levels and whether they are in the normal range. You should also be aware of how they affect you and what you can do to ensure they are functioning optimally.

Sermorelin acetate is a natural active analog of growth hormone releasing factor, (GHR) which is a growth hormone-releasing. (Sermorelin) Is naturally releaser to stimulate production healthily by the pituitary gland. Sermorelin Therapy is not a human growth hormone, it is a hormone secretagogue, which means that it stimulates the pituitary gland to specific receptors to increase the production of HGH.

Growth hormone-releasing peptides (GHRPs) are a family of synthetic peptides that stimulate GH secretion. Importantly, they have no structural similarity to sermorelin or GHRH; instead, they function via receptors in the pituitary gland and/or hypothalamus. At AAI Rejuvenation Clinic, we compound two different GHRPs (GHRP-2 and GHRP-6) into the sermorelin mixture. The resulting mixtures, sermorelin peptides GHRP2 and sermorelin peptides GHRP6, are particularly potent and effective GH-inducing agents that yield comparable results to typical yet considerably more expensive hGH therapy.

GHRP-2 is a synthetic analog of the peptide hormone ghrelin. Ghrelin is a small 28 amino acid peptide that is produced by the stomach in response to hunger. It also activates the growth hormone secretagogue receptor (also known as the ghrelin receptor) to stimulate GH production and metabolism. GHRP-2 functions similarly to stimulate the hypothalamus and increase the production of endogenous GH from the pituitary gland; synthesis then slowly drops back to baseline by the third hour after administration. This more closely replicates the pulsatile nature of endogenous GH production, which makes it advantageous and potentially safer compared with synthetic HGH use.

GHRP-6 is a hexapeptide synthetic analog of met-enkephalin, which is an endogenous opioid growth factor; however, GHRP-6 does not exhibit opioid activity. GHRP-6 works similarly to GHRP-2; however, it is a first-generation GHRP that does not induce GH levels as strongly as GHRP-2. Instead, it promotes food intake by stimulating hunger and helps increase energy metabolism; therefore, it is good for patients that need to increase their food intake.

Our interest is to help answer the question, what is human Growth Hormone, in as simple and concise a format while making sure no important details are missed. Simply put, Growth Hormone, as is encompassed in the name, literally helped to grow you; every aspect of you. When growth hormone is released, it is metabolized by the liver and converted into what is known as IGF-1 (Insulin Growth Factor-1). This chemical invites the production of protein in the body and depending on your different stages of development, promotes the growth of your muscles, bones as well as all your organs. In fact, Growth Hormone is responsible for the growth of all the tissue in your body. It promotes the regeneration and production of cells in the body, as well as stimulates their healing.

HGH is produced by top pharmaceutical organizations as a bio-identical hormone. This is identical to what the body produces with the exception that there is no DNA association. Administering hGH therapy in the not-so-distant-past was nothing to take lightly. It needed to be extracted from the brain of cadavers. This antiquated process didn’t come without its unfortunate repercussions. Patients treated with this early form of hGH had abstract side effects. This process is no longer legal. Today, technology has advanced to the point that we are able to clone and produce a clean, exact, bio-identical replica of the HGH hormone in a laboratory. When you qualify for therapy with AAI Rejuvenation Clinic, you will be prescribed Brand hGH only. HGH cannot be produced in a simple lab. It cannot be compounded or cannot be ingested, as the molecule is too delicate and small. It would be immediately destroyed by the gastric process. Contact us at AAI Clinics so we can talk to you about how to know if you are obtaining real hGH or, how to qualify for this life-transforming program.

 

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**NOTE**  The content contained in this blog is subject to interpretation and is the opinion of the content writer.  We do not claim it to be fact.  We encourage you to consult a medical doctor before taking any prescribed medications or supplements.

January 24, 2016 by admin 0 Comments

Hormone therapy must be with a real hormone specialist 5 (2)

Hormone Specialist Therapy

Hormone specialist therapy That is the point of our organization. Speak to an “actual”, specialized hormone specialist whom will help you understand what you will need in order to achieve all your health and fitness goals. Let us help you qualify for therapy. AAI Rejuvenation Clinic is a real, established Hormone Clinic in Fort Lauderdale, FL with an actual Bricks and Mortar location. Look us up. Fill out a form and a qualified Wellness Advisor will contact you with NO FURTHER OBLIGATION required on your part. Want to speak to the doctor directly? The very first “doctor” phone consult is free, even if you are not yet a patient of AAI Clinics. Fill out the form on our Medical History Form tab and someone will get back to you within 24 business hours.

Thank you for enjoying. To view the actual articles associated with these images click here.

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Why AAI Clinics, you ask yourself? AAI Rejuvenation Clinic offers an exciting new approach to anti-aging age management and total wellness for men and women. Utilizing the latest advances in total wellness we can help you look and feel your best. We believe in a comprehensive, individualized, scientific approach to wellness.

We partner with you to implement your wellness program. Our anti-aging treatments are custom-tailored to each individual because hormone therapy is not one-size fits all. Additionally, you will be assigned a Wellness Advisor to help guide you through your entire process.

We specialize in helping you revitalize your health and well-being while helping you feel more youthful. AAI Rejuvenation Clinic is operated by highly skilled, qualified professionals and physicians who focus on prescribing the proper anti-aging hormone therapy for each patient. As a result of our medical expertise, our specialists are exceptionally attentive to the details that make a difference in your health and wellness. Each of you is a unique individual; each of our treatments is customized to suit your personal needs and goals.

The process is simple. Once you’ve decided to take charge of improving your health and your future and you trust us to lead you in the process, you will reach out to us, either by clicking this Medical History Form link, or the tab above this page. Your information comes to us on a secure network. You can also call us at (866) 224-5698. We are here Monday – Friday 9am to 6pm EST.

November 16, 2015 by Joseph Fermin 0 Comments

Sermorelin Peptides GHRP and Benefits of HGH 5 (3)

Sermorelin Peptides GHRP and Benefits of HGH

Sermorelin peptides GHRP and benefits of hGH are undeniable. However, when using injectable exogenous HGH (GH not produced naturally by our body) our bodies go from producing low amounts of GH to essentially producing no GH. The reason this occurs is that the body becomes completely dependent on the exogenous hGH. As incredible as it is to have higher levels of GH in your body, it’s extremely important to cater to the health of your pituitary gland to ensure long-term health and positive results. So how do you get your pituitary gland to start working again? More importantly, how do you get it to work harder and more efficiently for an individual your age or with your particular abnormal pituitary condition? This is a job for Sermorelin peptide GHRP peptides.

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Sermorelin

  • Sermorelin peptides (GHRH) is a bio-identical hormone that has recently been genetically engineered to stimulate the secretion of Growth Hormone releasing hormone from the hypothalamus. GHRH is a peptide that contains the first 29 amino acids of the 191 in GH. It is GHRH that stimulates your pituitary gland to release GH. As we age, hormone production in the pituitary gland declines. It has been proven that HGRH can restore the production levels of the pituitary causing significant elevation of IGF-1.

Peptides

  1. One of the major functions of peptides is to work as hormones because these are a specific type of peptide responsible for carrying signals between cells and glands. These peptides will control blood sugar regulation, sleep, and other body functions. They are created by the liver, intestine, stomach, kidneys and endocrine glands. Common peptides found in hormones include oxytocins, gastrins, bradykinins, human growth hormone and insulin.
  2. – The brain produces neuropeptides, which you find in neural tissue. They are an extension of your neuro system throughout your body, meaning that they act as regulators and signals in various processes triggered from locations within the brain. One example of neuropeptides would be endorphins.
  3. – Alkaloids are another type of peptide and they are commonly involved in developing defense mechanisms in small animals, plants, and fungi. Examples of these peptides include dynorphin, panda mini, and ergotamine.
  4. – When peptides work as antibiotics, they prevent microorganisms from growing within the body. This is an example of using peptides side effects to kill bacteria that cause diseases as antibiotics are commonly used in medicine.
  5. – The peptide can help support our body. This is because when peptide chains become long enough, it will form proteins, which are used as building blocks of animal and human bodies. Keratin is a perfect example as this protein is in human nails and hair and animal claws, hooves, scales, feathers, and horns. Collagen is another protein created from peptides and is found in bones, tooth enamel, ligaments, and tendons.

GHRP’s

  • GHRP’s are a small family of peptides acting in the hypophyseal portal system to release Growth Hormone (GH). There are two main forms of GHRP, which will be depicted further below, that are compounded into the Sermorelin Peptides GHRP mixture. The end result, Sermorelin GHRP2 or Sermorelin GHRP6, is an especially potent and effective Growth Hormone inducing agent that can generate comparable results to a typical, yet considerably more costly, hGH therapy protocol.

GHRP2

  • Growth-Hormone-Releasing-Peptide-Two (GHRP2) is a synthetic ghrelin analogue. It stimulates the hypothalamus and critically increases the natural production of Growth Hormone (GH) from the pituitary gland, which slowly drops back to baseline levels by the third hour after administration. This more closely replicates the natural pulse of GH in the human body, giving it an advantage over synthetic HGH use

– GHRP-2 (also known as KP 102) is a synthetic hexapeptide Growth Hormone Releasing Peptide (GHRP) which acts on the hypothalamus and the pituitary gland to release growth hormone with a slight stimulatory effect on Prolactin, ACTH, and Cortisol levels.

– GHRP-2 is considered to be a true hGH secretagogue, meaning that it stimulates the body’s own secretion of hGH. Human Growth hormone has been shown in studieshttps://www.aaiclinics.com/treatments/hgh-therapy/what-is-human-growth-hormone/ to promote lean body mass and reduce adiposity (fat).

– GHRP2 has demonstrated that it is very effective at stimulating GH production in research test subjects.

– It has a short half-life with peak concentrations occurring around 15 minutes and not longer than 60 minutes after administration.

– GHRP-2 is a 2nd generation GHRP just behind GHRP-6. Compared to GHRP-6, GHRP-2 is considered to be much more superior in terms of growth hormone stimulation because over longer periods of time it maintains maximum elevations in Growth Hormone.

– GHRP-2 has shown to increase IGF-1 levels (Insulin-like Growth Factor-1), and even greater results happen when used with Sermorelin Peptides GHRP (Growth Hormone Releasing Hormone – GHRH). It has a short half-life with peak concentrations occurring around 15 minutes but not longer than 60 minutes. The response of natural physiologic system includes the increase in levels of calcium ion influx alongside with increased release of growth hormones in response to the high-amplitude pulsation from the GHRP-2.

– GHRP-2 and other ghrelin analogues increase the number of somatotropes involved in the GH pulse by inhibiting somatostatin, GHRH increases the pulse amplitude per pituitary cell or somatotrope by other means.

-Unlike ghrelin, GHRP-2 is not lipogenic which means that it does not induce fat storage. Although ghrelin plays a large role in hunger, GHRP-2 as an analog of ghrelin does not increase appetite significantly.

GHRP6

  • Growth-Hormone-Releasing-Peptide-Six (GHRP6) is a Growth Hormone (GH) secretagogue. The Peptide GHRP-6 works similarly to GHRP-2, however, it is a Hexapeptide that promotes food intake by stimulating hunger and helps increase energy metabolism.

Growth Hormone Releasing Peptides, similar to GHRP-6, are most commonly used for the treatment of Growth Hormone (GH) deficiencies, eating disorders, etc.

– Researchers discovered that those GH-Releasing Peptides (specifically GHRP-6 & GHRP-2) followed a mode of action, which bound them to and was mediated through receptors different from those for GHRH.

– Researchers discovered that these GH-Releasing Peptides acted synergistically with the natural human Growth Hormone Releasing Hormone (GHRH), which is related to Sermorelin Peptides GHRP, in both laboratory animals and humans to produce large releases of Growth Hormone.

– In the 1980s, the first highly potent GH-Releasing peptide, GHRP-6, was developed. Due to a strong GH release response from the peptide, it became the first member of a class called Growth Hormone Secretagogues.

– GHRP-6 is believed to be acting naturally on both pituitary and hypothalamic sites (Fairhall et al. 1995). In a time-dependent and dose-dependent manner, the primary pituitary cells of rats were demonstrated on.

  • Both GHRP-2 and GHRP-6 have great benefit and potential. Studies have shown them to be effective in treating age-related GH decline when used in combination with Sermorelin (GHRH).

GHRP’s when used in equal dosages of GHRH creates a 3-hour pulse of GH that is double the amplitude of 8IU of synthetic GH, resulting in:

a) increased energy

b) improved sleep

c) increased lean body mass

d) decreased body fat

e) increased collagen production and

f) increased healing capability.

These factors not only make it more effective but actually a healthier substitute to Exogenous Growth Hormone.

Overview: Sermorelin Peptides,
Sermorelin GHRP2 & 6 Peptides

Use of Sermorelin usually improves sleep within the first few weeks of therapy. This is usually concurrent with increased energy levels and improved mood. After 3-6 months of therapy, patients start reporting noticeable or significant body changes, such as the increase in muscle tone and leaner physique. Over time patients will also notice a significant improvement in skin tone and health. Like most peptide hormones, Sermorelin Peptides GHRP usually has a loading period of 3-6 months before full effects are noticed. Once injected, both Sermorelin Peptides GHRP are eliminated very quickly from the body and therefore need to be injected frequently. Its actions are dependent on a chain reaction of biological processes which result in elevated and sustained HGH and growth factors.

When considering Growth Hormone Therapy, personal preference and disposition are always the determining factors. However, Sermorelin Peptides GHRPs is a wonderful option to getting you back your vitality and youth.

Contact us at your earliest convenience so we can give you more information about how HGH therapy and/or Sermorelin therapy can make a difference in your health. We look forward to helping you decide which replacement therapy is best for you. AAI Rejuvenation Clinic’s anti-aging professionals are your resource source to extend your life and get more from it.

Sermorelin Injection – Curious about Benefits of Sermorelin Therapy? Read more about what you can expect from this treatment and contact us for more information (866) 224-5698

WHAT IS SERMORELIN?

September 20, 2015 by Joseph Fermin 0 Comments

SERMORELIN GHRP 2 5 (1)

SERMORELIN GHRP 2

GHRP 2 is a Growth Hormone Releasing Peptide Two (GHRP2) is a synthetic ghrelin secretagogue. It stimulates the hypothalamus and critically increases the natural production of Growth Hormone (GH) from the pituitary gland, which slowly drops back to baseline levels by the third hour after administration. This more closely replicates the natural pulse of GH in the human body, giving it an advantage over synthetic hGH use.

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  • GHRP-2 (also known as KP 102) is a synthetic hexapeptide Growth Hormone Releasing Peptide (GHRP) which acts on the hypothalamus and the pituitary gland to release growth hormone with a slight stimulatory effect on Prolactin, ACTH, and Cortisol levels.
  • GHRP-2 is considered to be a true hGH secretagogue, meaning that it stimulates the body’s own secretion of hGH. Human Growth hormone has been shown in studies to promote lean body mass and reduce adiposity (fat).
  • GHRP2 has demonstrated that it is very effective at stimulating GH production in research test subjects.
  • It has a short half-life with peak concentrations occurring around 15 minutes and not longer than 60 minutes after administration.
  • GHRP-2 is a 2nd generation GHRP just behind GHRP-6. Compared to GHRP-6, GHRP-2 is considered to be much more superior in terms of growth hormone stimulation because over longer periods of time it maintains maximum elevations in Growth Hormone.
  • GHRP-2 has shown to increase IGF-1 levels (Insulin-like Growth Factor-1), and even greater results happen when used with Sermorelin (Growth Hormone Releasing Hormone – GHRH). It has a short half-life with peak concentrations occurring around 15 minutes but not longer than 60 minutes. The response of natural physiologic system includes the increase in levels of calcium ion influx alongside with increased release of growth hormones in response to the high-amplitude pulsation from the GHRP-2.
  •  GHRP-2 and other ghrelin secretagogue increase the number of somatotropes involved in the GH pulse by inhibiting somatostatin, GHRH increases the pulse amplitude per pituitary cell or somatotrope by other means.
  • Unlike ghrelin, GHRP-2 is not lipogenic which means that it does not induce fat storage. Although ghrelin plays a large role in hunger, GHRP-2 as a secretagogue ghrelin does not increase appetite significantly.

Sermorelin Injection – Curious about Benefits of Sermorelin Therapy? Read more about what you can expect from this treatment and contact us for more information (866) 224-5698

WHAT IS SERMORELIN?

**NOTE**  The content contained in this blog is subject to interpretation and is the opinion of the content writer.  We do not claim it to be fact.  We encourage you to consult a medical doctor before taking any prescribed medications or supplements.

September 20, 2015 by Joseph Fermin 0 Comments

SERMORELIN GHRP 6 5 (1)

SERMORELIN GHRP 6

GHRP 6 Growth Hormone Releasing Peptide Six (GHRP6) is a Growth Hormone (GH) secretagogue. The Peptide GHRP-6 works similarly to GHRP-2, however, it is a Hexapeptide that promotes food intake by stimulating hunger and helps increase energy metabolism.

  • Growth Hormone Releasing Peptides, similar to GHRP-6, are most commonly used for the treatment of Growth Hormone (GH) deficiencies, eating disorders, etc.
  • Researchers discovered that those GH-Releasing Peptides (specifically GHRP-6GHRP-2) followed a mode of action, which bound them to and was mediated through receptors different from those for GHRH.
  • Researchers discovered that these GH-Releasing Peptides acted synergistically with the natural hormone Growth Hormone Releasing Hormone (GHRH), which is related to Sermorelin, in both laboratory animals and humans to produce large releases of Growth Hormone.
  • In the 1980s, the first highly potent GH-Releasing peptide, GHRP-6, was developed. Due to a strong GH release response from the peptide, it became the first member of a class called Growth Hormone secretagogues.
  • GHRP-6 is believed to be acting naturally on both pituitary and hypothalamic sites (Fairhall et al. 1995). In a time-dependent and dose-dependent manner, the primary pituitary cells of rats were demonstrated on.

Both GHRP-2 and GHRP-6 have great benefit and potential. Studies have shown them to be effective in treating age-related GH decline when used in combination with Sermorelin (GHRH).

  • GHRP’s when used in equal dosages of GHRH creates a 3-hour pulse of GH that is double the amplitude of 8IU of synthetic GH, resulting in:

a) increased energy

b) improved sleep

c) increased lean body mass

d) decreased body fat

e) increased collagen production and

f) increased healing capability.

These factors not only make it more effective but actually a healthier substitute to exogenous Growth Hormone.

If you have any questions or comments related to this information or if you are interested in more details about the requirements for therapy qualification, don’t hesitate to fill out our Medical History Form. Once you do, you will be contacted within 24 business hours by one of our friendly wellness advisors.

Sermorelin Injection – Curious about Benefits of Sermorelin Therapy? Read more about what you can expect from this treatment and contact us for more information (866) 224-5698

WHAT IS SERMORELIN?

**NOTE**  The content contained in this blog is subject to interpretation and is the opinion of the content writer.  We do not claim it to be fact.  We encourage you to consult a medical doctor before taking any prescribed medications or supplements.

September 20, 2015 by Joseph Fermin 0 Comments

The Protocol Sermorelin Therapy vs HGH Therapy 5 (1)

Protocol Sermorelin Therapy

Protocol Sermorelin therapy, We reiterate that there is no one-protocol-fits-all when it comes to any kind of hormone therapy. Be warned: if you are speaking to an organization that is offering you solid quotes before their doctors have had an opportunity to review your file details, you should be very weary. It doesn’t mean that they cannot legally obtain medication for you. It simply implies your health is not at the forefront of that transaction. In fact, it’s most possible, that is all your communication is to that organization; a transaction.

Your protocol needs to be individualized based on your complete health history and file characteristics. That being said, there are certain similarities from one protocol to the next and certainly, there are some particular characteristics that all subcutaneous protocols have in common.

The following information is not to be used in substitution or in combination with anything that your prescribing physician has organized for you. The information below is only to be regarded as educational and generally suggestive when relating to subcutaneous Protocol Sermorelin therapy. When you have been prescribed a Sermorelin or Growth Hormone program, your doctor should explain to you:

  • the areas where you will inject the medication
  • exact time and day you should be administering your medicine
  • how to handle and store the medication
  • information about needles and all pertinent supplies in order to properly and effectively administer your program.

Other important points to consider when on a physician supervised Sermorelin protocol:

  • You must not eat or drink anything but water 90 minutes before or after your Protocol Sermorelin Therapy administration. Sermorelin impacts your insulin levels. Food does the same thing. Eating food in close proximity to your Sermorelin administration will surely null and void that injection.
  • Sermorelin should not be administered during the day. The reaction of Sermorelin to effectively focus and work on the pituitary, hypothalamus, serotonin levels and related areas of the brain actually promotes a sleepy reaction that can be inhibiting for participants during the day. Because Sermorelin so actively works on the pituitary to stimulate secretion, it induces a sense for needed rest that is difficult to combat.
  • Sermorelin is known to stimulate hunger in an effort to produce the resources and energy necessary to effectively metabolize your produced GH into IGF-1 which is needed to promote the protein synthesis required to generate the benefits expected from a GH therapy.
  • Sermorelin should be injected subcutaneously. Your best option is to administer Sermorelin directly into fat. Injecting Sermorelin into muscle will cause the medication to be hyper-metabolized. If you inject it into fat, it is processed much more slowly, extending the half-life of your administration. Conversely, if you inject Sermorelin directly into your muscle, it is much more quickly metabolized and processed by the body so its functionality/effectiveness time is reduced.
  • Because Sermorelin serves as a stimulant to the pituitary and hypothalamus, it actually helps to regenerate and rejuvenate the glands, promoting their health and efficiency. For this reason, it is safe and recommended to administer Sermorelin on a 7-night administration weekly cycle. Because Sermorelin is inducing production of your own GH and it’s forcing maximum secretion of your involved glands, it’s best to be very consistent with this therapy, especially when aiming to achieve results reflective of a much more expensive, straight bio-identical HGH protocol.

The protocol for Sermorelin is really a simple one. Retrospectively, despite Sermorelin also being a Growth Hormone therapy, promoting the same results as an HGH program in participants with a well-functioning pituitary, they administration times are quite opposite.

Remember:

  • HGH must be administered in the day-time. The intention is to inject this medication when it will least interfere with the body’s natural production of Growth Hormone. Our endocrine system is most active at nighttime and this holds particularly true for the secretion period for Growth Hormone. Growth Hormone is released during several different phases of the day. However, the majority of the Growth Hormone your body is going to benefit from is released at night, during REM sleep (deepest, most restful sleep stage). If you administer hGH at night time, it will interfere with your natural production of GH, as your body checks all your blood levels constantly to determine if any given secretion is required to normalize and maintain normal bodily function. When the body detects that GH levels are normal (resulting from the HGH administration), it will signal the brain to not produce any additional GH. Extended hGH therapy on this improper protocol will most certainly cause atrophy of the pituitary from lack of use.
  • Conversely, Protocol Sermorelin Therapy does not promote an artificial elevation of GH levels. As a secretagogue, it actually induces your own production of Growth Hormone. The GH surge in your body is all-natural; your own. It is not a man-made or bio-identical hormone. It is real, DNA associated Growth Hormone secreted by the pituitary and hypothalamus as stimulated by the administration of Sermorelin. For this very reason, the opposite is true regarding the appropriate time to administer protocol Sermorelin therapy on your required administration days.
  • The smartest thing any GH therapy participant could do is consider a complete, well-rounded protocol inclusive of both Sermorelin and HGH at the same time. Of course, the dosages would be tweaked to ensure there isn’t an exaggerated overall dose of GH for the body to deal with at any one given time. Depending on the patient’s goals and file details, the Sermorelin/hGH combination therapy can be structured in a wide variety of effective ways. Combining HGH and Sermorelin is also a significantly effective way to participate in a leveler impacting the program like hGH without having to deal with the much higher costs of an only HGH hormone replacement program.

Typically, what dosage would somebody of my height, weight, and age be prescribed for HGH therapy?

 It’s imperative to understand that no one should ever generalize a program for you. It’s crucial that your prescribing physician takes into account all of your file details, health history, and personal goals before structuring a Sermorelin protocol, as we will do for you here at AAI Rejuvenation Clinic.

Beyond that clarification, statistically:

  • A person weighing between 100lbs -140lbs, between 4.5 – 5ft tall, would be prescribed 200mcg of Sermorelin every evening before bed.
  • A person weighing between 141lbs -165lbs, between 5 – 5.8ft tall, would be prescribed 250mcg of Sermorelin every evening before bed.
  • A person weighing between 166lbs -195lbs, between 5.5 – 6ft tall, would be prescribed 250 – 300mcg of Sermorelin every evening before bed.
  • A person weighing between 196lbs – more, between 5.5 – or taller, would typically be prescribed 300mcg or more, as determined by the physician, of Sermorelin every evening before bed.

Side Note: Elderly people and women will be prescribed higher dosages than outlined above to stimulate the kind of GH production needed to achieve the desired benefits and results depicted in these educational sites.

Combination HGH and Sermorelin Protocol

The more common ‘combination protocols will significantly very in dosing, as this is an art your prescribing physician needs to be well acquainted with in order to promote safe and effective results while keeping unwanted side effects at bay. At AAI Rejuvenation Clinic, our physicians are experts in the field of hormone replacement therapy and qualified by the Anti-Aging Association board.

A program can look something like this:

For someone that is very receptive to therapy, that doesn’t need as much, they may only need Sermorelin on the weekend.

  • Sat and Sun.

This ensures they are getting a healthy dose of GH elevation 7 days a week without putting the health of their pituitary at stake.

  • There is also the case in which your physician, in an effort to maximize your IGF-1 levels to expedite the physiological response to therapy whilst still keeping unwanted side effects at bay, may significantly reduce your hGH administration in the morning  but intelligently substitute the additional GH supplementation through the introduction of natural Growth Hormone via the selected AAI physician secretagogue, either Sermorelin GHRP2 or Sermorelin GHRP6, every evening in addition. Usually, still, hGH is not administered on a 7 consecutive day basis.

Patients needing the benefits of Growth Hormone that wish to participate in hGH therapy but are restricted by the costs may consider a combination protocol that entails either a much lower dosage of hGH during the day (allowing them to need much fewer vials of the hGH to complete their program) while substituting their GH elevation requirements for the day by introducing Protocol Sermorelin Therapy in the evening as well. This not only assists in providing an effective and potent GH enriching program but, at the same time, it’s a protocol that strategically caters to the health of the pituitary by inducing its work to secrete GH regardless of the current levels due to therapy. This ensures the participant of this protocol never has to worry about atrophy of the pituitary. In fact, this participant can rest assured they are on a well-rounded program that is intended to maximize their health benefits while minimizing any unwanted side effects.

Again, we reiterate that the details above are statistically depicted and there is no chart for dosing an effective and safe Sermorelin protocol. However, the above chart most accurately represents, statistically, what a patient of a certain height, age and weight can expect to be prescribed for a safe and effective Protocol Sermorelin therapy program.

Sermorelin Injection – Curious about Benefits of Sermorelin Therapy? Read more about what you can expect from this treatment and contact us for more information (866) 224-5698

WHAT IS SERMORELIN?

**NOTE**  The content contained in this blog is subject to interpretation and is the opinion of the content writer.  We do not claim it to be fact.  We encourage you to consult a medical doctor before taking any prescribed medications or supplements.