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Category: BMI

March 5, 2019 by Joseph Fermin 0 Comments

What is Follicle Stimulating Hormone (FSH)? 5 (8)

What is Follicle Stimulating Hormone

What is Follicle Stimulating Hormone (FSH), is one of the gonadotrophic hormones, and the other being a Luteinizing Hormone (LH). The pituitary gland releases both into the bloodstream and body, and Follicle Stimulating Hormone (FSH) is one of the hormones essential for the development function of women’s ovaries and men’s testes. In women, Follicle Stimulating Hormone (FSH) stimulates the growth in the ovary before the release of an egg from one follicle to the ovulation. It also increases estradiol production. In men, Stimulating Follicle Hormone (FSH) acts on the Sertoli cells of the testes to stimulate sperm production (spermatogenesis).

How is Follicle Stimulating Hormone (FSH) control?

The release of Follicle Stimulating Hormone (FSH) is regulated by the levels of some circulating hormones released by the ovaries and testes. This system is called the hypothalamic–pituitary–gonadal axis. The gonadotropin-releasing hormone is published in the hypothalamus and the receptors in the anterior pituitary gland to stimulate both the synthesis release of Follicle Stimulating Hormone (FSH) and Luteinizing Hormone (LH). The released Follicle Stimulating Hormone (FSH) is carried in the bloodstream, where it binds to receptors in the testes and the ovaries. Using this mechanism Follicle Stimulating Hormone (FSH), along with Luteinizing Hormone (LH), can control the functions of the ovaries and testes.

follicle stimulating hormone

In women, when hormone levels are deficient, and it has complication the menstrual cycle, this is sensed by nerve cells in the hypothalamus. These cells produce the more gonadotrophin-releasing hormone, which in turn stimulates the pituitary gland to produce more Follicle Stimulating Hormone (FSH) and Luteinizing Hormone (LH), and release these into the bloodstream. The rise in Follicle Stimulating Hormone (FSH) and stimulates the growth of the follicle in the ovary, and the cells of the follicles produce increasing amounts of estradiol. In turn, this production of these hormones is sensed by the hypothalamus, and pituitary gland and less gonadotrophin-releasing hormone and Follicle Stimulating Hormone (FSH) will be released, However, as the follicle grows, and more and more estrogen is produced from the follicles, it simulates a surge in luteinizing hormone (LH) and Follicle Stimulating Hormone (FSH), which stimulates the released egg from a mature follicle – ovary.

During women menstrual cycle, there is a rise the Follicle Stimulating Hormone (FSH) secretion in the first half of the period and stimulates follicular growth in the ovary, after ovulation, each month the ruptured follicle forms and Corpus luteum that produces high levels of progesterone. This inhibits the release of stimulating Follicle Stimulating Hormone (FSH), and towards the end of the cycle the Corpus luteum breaks down, and progesterone production decreases. The next menstrual period begins when Follicle Stimulating Hormone (FSH) starts the production again, and get back to normal…

follicle stimulating hormone

Now In men, the production of Follicle Stimulating Hormone (FSH) is regulated by levels of testosterone and inhibin, both produced by the testes. Follicle Stimulating Hormone (FSH) regulates testosterone levels and when this rise they are sensed by nerve cells in the hypothalamus so that gonadotropin-releasing hormone secretion and consequently Follicle Stimulating Hormone (FSH) is decreased. The opposite occurs when testosterone levels drop. This is known as a ‘Negative Feedback in the body’ control so that the production of testosterone remains steady. But the sensed by cells in the anterior pituitary gland rather than the hypothalamus.

What happens if you have too much Follicle Stimulating Hormone (FSH)?

Most often, and raised levels of Follicle Stimulating Hormone (FSH) are a sign of malfunction in the ovary or testis. If the gonads fail to create enough estrogen, testosterone and inhibit, the right feedback control of Follicle Stimulating Hormone (FSH) production from the pituitary gland is lost, and the levels of both Follicle Stimulating Hormone (FSH) will rise. This condition is called hypogonadotropic-hypogonadism and is associated with primary ovarian failure or testicular failure. This is seen in states such as Klinefelter’s syndrome in men and Turner syndrome in women.

In women, Follicle Stimulating Hormone (FSH) levels also start to rise naturally in women around the menopausal period, reflecting a reduction in the function of the ovaries and decline of estrogen and progesterone production.

There are rare pituitary conditions that can raise the levels of Follicle Stimulating Hormone (FSH) in the bloodstream. This overwhelms the regular negative feedback and can cause ovarian hyperstimulation syndrome in women ovaries.

Symptoms: This includes enlarging of the ovaries and potentially dangerous accumulation of fluid in the abdomen, and triggered the rise in ovarian steroid output. Which leads to pain and other problems in the pelvic area of the body.

What happens if don’t produce enough Follicle Stimulating Hormone (FSH)?

In women, lack of Follicle-Stimulating-Hormone (FSH) leads to incomplete development in puberty o poor ovarian function (ovarian failure), and In this situation ovarian follicles do not grow properly and do not release in the egg, thus leading to infertility. Since levels of Follicle-Stimulating-Hormone (FSH) in the bloodstream are low, this condition is called hypogonadotropic-hypogonadism. This condition is called Kallman’s syndrome, which is associated with a reduced sense of smell.

Sufficient Follicle-Stimulating-Hormone (FSH), this action is also needed for proper sperm production in men, in case of complete absence of Follicle Stimulating in men, and the lack of puberty and infertility due no production of sperm is called (azoospermia). Partial Follicle-Stimulating-Hormone (FSH) deficiency in young men, can also cause delayed puberty and low sperm production, called (oligozoospermia), but fathering a child may still be possible. Follicle-Stimulating-Hormone (FSH) occurs after puberty; there will be a similar loss of fertility…

January 28, 2019 by Joseph Fermin 0 Comments

The importance of Testosterone Post Cycle Therapy? 5 (5)

Why Is Testosterone
Post Cycle Therapy Is Need It?

Why is Post Cycle Therapy (PCT) is perhaps the most critical aspect of testosterone use? The concept of the post cycle therapy (PCT), did not exist before the late 1980s, and 1990s and the mechanisms by which testosterone affected, the body were not wholly understood during the 1950s, 1960s, and 1970s.

This period were doctors, scientists, and testosterone injections users were only beginning to learn about the dynamics of testosterone and how they affect the endocrine system. We believed and understood since the beginning of testosterone injections use, the administration of testosterone resulted in triggering the body’s negative loop of the (HPTA) Hypothalamic Pituitary Testicular Axis. That endogenous Testosterone production would result become suppressed and shut down. The, unfortunately, is during the early periods of testosterone use between the 1950s and 1990, there was limited access to the compounds or knowledge or effectively.

Today it is a very different story. Now scientific and medical understanding of bio-identical testosterone use has soared exponentially since the old ‘golden era’ days of looking young and testosterone therapy use in athletics. Countless developments of beneficial compounds for hormonal recovery after testosterone therapy use, alongside the increased scientific and medical knowledge, has enabled testosterone use and its associated endocrine disruptions. The proper knowledge on how to recover the body’s from Hypothalamic Pituitary Testicular Axis (HPTA). Through post cycle therapy (PCT), we can not only emerge from their testosterone therapy while holding on to almost all of their benefits, but they can also increase the chances upwards to 90 percent or higher range of emerging with a fully healthy (HPTA).

Following the use of exogenous testosterone injections, the majority of users will experience what has been a hormonal crash or post cycle therapy crash, which is a physical environment in which key hormones essential is has been suppressed or shut down. The critical hormones in question are Luteinizing Hormone (LH), Follicle Stimulating Hormone (FSH), and subsequently (and are most importantly), for our natural testosterone. The Luteinizing Hormone (LH), and the Follicle Stimulating Hormone (FSH), known as phototropism. These hormones increase Testosterone secretion. Also alongside low levels of these hormones, to balance the essential hormones that have been thrown off balance, whereby Testosterone levels will be small, and most of the time, depending on the factors, estrogen levels will be higher than usual, and levels of Cortisol a steroid hormone that destroys muscle tissue. With the testosterone levels low and Cortisol levels in the average or high, Cortisol now can become a threat to the new muscle during the new testosterone therapy (“Testosterone correctly suppresses and counteracts Cortisol’s catabolic effects on muscle tissue”). The SHBG (Sex Hormone Binding Globulin) is also a concern here as well, which is a protein that binds to sex hormones Testosterone renders them inactive, essentially ‘handcuffing’ them and preventing them from exerting their effects. SHBG will also usually elevated during the post cycle therapy weeks as a result of the supraphysiological levels of androgens from the new testosterone therapy.

The human body will generally and restore this imbalance of hormones and recover from testosterone levels on its own, over time with no outside assistance or post cycle therapy (PCT), but the studies have demonstrated and shown us that without the intervention of testosterone stimulating agents, this will occur throughout one to four months. Therefore, all testosterone therapy should be concerned with the fastest possible hormonal recovery, assisted and boosted with the use of Testosterone stimulating compounds correctly, also the attempt to allow the body to recover on its own, from a very high probability of long-term endocrine damage to the Hypothalamic Pituitary Testicular Axis (HPTA), whereby the individual will develop-induced hypogonadism to inability the production of proper levels of Testosterone to rest. So therefore paramount that an appropriate post cycle therapy that includes multiple recovery compounds to be utilized to not only restore the (HPTA) function but also to normalize the levels as quickly as possible. To avoid any possible permanent damage, which can take priority over the concern of maintain to the recently gained muscle mass and any other benefits from it.

What Post Cycle Therapy Protocol?

There are many different types of post cycle therapy (PCT) protocols that have overdeveloped over the years; any individual will become extremely confused about how many different opinions exist among the testosterone community, This article will present the best possible and most efficient post cycle therapy protocol valid scientific data, also myths in regards to post cycle therapy (PCT), and outline which post cycle therapy (PCT) protocols should not follow due to recent more advanced developments, as well as contemporary better scientific and medical understandings of how a proper post cycle therapy protocol should work. This point, there still exists very obsolete – and subsequently ineffective – post cycle therapy (PCT) contracts that are still utilized by many testosterone users, and this presents a severe hazard not only for the individual unknowingly using a post cycle therapy.

For example:

There are several therapeutic and safety reasons why you should not continue with testosterone injections indefinitely without giving your body time to normalize to reset. Because of the decline in benefits after six months of a testosterone therapy, the physicians need to regularly incorporate a cleanse therapy post cycle therapy (PCT) in an attempt to reactivate the endocrine in the body, as you increase your testosterone levels using any testosterone therapy, now the levels of testosterone circulating the body will shut down the natural production of your endogenous testosterone; and also increases the production of estrogen in your body, which can lead to a series of undesirable and unwanted side effects in the body. `This means that the synthesis of (LH) luteinizing hormone in your body; this hormone is produced by your brain to stimulate testosterone production. and follicle-stimulating hormone in the body; the hormone produced by your mind to boost sperm production suddenly stops. When Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) levels are no longer detectable, your body will not experience the exceptional health benefits, and energy-optimizing results expected from a testosterone injection program.

Another critical concern for men is testicular atrophy in patients that participate in testosterone therapy and may experience shrinkage of the testes, This occurs as the result of the lack of testosterone, and sperm production has been shut down, in response to the testosterone therapy.

What does post cycle therapy (PCT) consist of, an example?

Your post cycle therapy consists of a testosterone secretagogue to stimulate the secretion of endogenous testosterone from the testes to reignite natural production. The medication mimics the signal from your brain. The Luteinizing Hormone (LH) induce the production of testosterone. An example of a testosterone secretagogue is human chorionic gonadotropin (hCG), which is administered either using sublingual troches or subcutaneous injections once or twice a week during therapy and then on 10–15 consecutive days as part of a post cycle therapy (PCT). Human chorionic gonadotropin (hCG), mimics Luteinizing Hormone (LH) to stimulate testosterone production by the testes. It works by effectively tricking the testes into thinking that they are being instructed to produce testosterone, even though levels are comfortably elevated because of the injectable testosterone therapy. The testosterone production stimulated by human chorionic gonadotropin (hCG) is not sufficient to sustain healthy testosterone levels on its own, but that is not the reason for this supplementation. The purpose is to ensure that the testes remain functioning during therapy to help avoid any shrinkage or atrophy.

You will also take an anti-estrogen or aromatase inhibitor. For example, Clomid/clomiphene blocks certain types of estrogen from getting to the pituitary and hypothalamus, where it elicits signals that stop testosterone production. Anti-estrogens or aromatase inhibitors also help to reactivate the standard functionality of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) signaling, while also helping to flush out any residual estrogen that has accumulated during therapy. The estrogen that collects during treatment is responsible for many of the adverse side effects associated with testosterone therapy.

An example of a post cycle therapy (PCT) protocol is as follows (note that the exact drug and dose prescribed will depend on the specific information contained within each patient file, as well as the individual patient goals):

  • 250–800 units of a testosterone secretagogue every day for ten consecutive days
  • One estrogen blocker or antagonist by mouth every day for 10–15 straight days

Without the proper understanding of what is explicitly occurring within the endocrine system during these crucial weeks, as well as a lack of knowledge of which compounds to utilize, what each compound does, and how to properly use them, serious problems can result.

post cycle therapy

The Hypothalamic Pituitary Testicular Axis (HPTA):

The (HPTA), which is an axis interconnected endocrine glands in the body that deals with control the production Testosterone.

Post-Cycle-therapy:

Outlined above is a diagram of the Hypothalamic Pituitary Testicular Axis (HPTA), Regulates the body produces the amount of Testosterone at any given time. Every individual is essentially programmed by (DNA) genetics as to maximum Testosterone they will provide.

The Hypothalamic Pituitary Testicular Axis (HPTA) and the functions that undergo a negative feedback loop, and the body will reduce secretion of Testosterone, f have too much Testosterone the body will be detected, known as the negative feedback loop. This controlled by the hypothalamus, which is mostly considered the ‘master’ gland for all endocrine system and the hormonal functions in the body. The negative feedback will loop ultimately in the body to attempt to maintain the hormonal homeostasis, and all endocrine glands operate by way of the negative feedback loop in one way or another in varying degrees, In the case of post cycle therapy, the concern is a negative feedback loop of the (HPTA).

Within the Hypothalamic Pituitary Testicular Axis (HPTA), the concern during post cycle therapy (PCT) is the restoration and regulation of the following five hormones to homeostasis:

  • GnRH (Gonadotropin Releasing Hormone)
  • LH (Luteinizing Hormone)
  • FSH (Follicle Stimulating Hormone)
  • Testosterone production

The Hypothalamic Pituitary Testicular Axis (HPTA), the hypothalamus, which will detect a need for the human body to produce more Testosterone, and will release varying amounts of GnRH, Is a hormone that signals the pituitary gland, to begin the production and release of two essential gonadotropins: Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH). Two hormones that work together to start the secretion of Testosterone.

Two primary hormonal factors serve to inhibit, reduce, suppress, or shut down Testosterone production in the Hypothalamic Pituitary Testicular Axis (HPTA):

  • Testosterone Excess
  • Estrogen Excess

Although there exist other hormones that serve to inhibit and suppress Hypothalamic Pituitary Testicular Axis (HPTA) function (such as Progestins and Prolactin), these are the two primary conditional hormones that are of concern. When the hypothalamus detects excess levels of Testosterone and Estrogen in the body (either from the use of exogenous androgens on an testosterone therapy or otherwise), the hypothalamus will act to attempt to restore a balance by essentially doing the opposite of what was previously described. The hypothalamus will reduce or stop its production of GnRH, which halts production of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH), which ultimately reduces or halts production of Testosterone. Until the hypothalamus’ ideal hormonal environment is restored, the output of the various signaling hormones within the (HPTA) will not begin, and this will often require months for the body to do this on its own without the intervention of any Testosterone stimulating agents. The reason as to why the recovery of the (HPTA) naturally takes such a long time should be very clear due to the described workings of the (HPTA).

This fundamental understanding of the mechanisms of the Hypothalamic Pituitary Testicular Axis (HPTA) and negative feedback loop described above is essential to understanding how and why a proper post cycle therapy (PCT) program must be developed and utilized following an testosterone therapy.

Determining Factors In Difficulty Recovering the Hypothalamic Pituitary Testicular Axis (HPTA):

With testosterone therapy use, there are several different major determining factors in how much difficulty an individual will experience in recovery of their Hypothalamic Pituitary Testicular Axis (HPTA) and endogenous Testosterone function during post cycle therapy (PCT).

They are the following factors, in no particular order of importance:

  • Individual response
  • Type of testosterone(s) used
  • Length of the cycle (degree of testicular desensitization)

Individual response:

Every single individual will respond differently to any chemical, compound, testosterone, food or drug in existence. While some individuals might experience no Hypothalamic Pituitary Testicular Axis (HPTA) suppression or shutdown at all, other individuals might experience severe Hypothalamic Pituitary Testicular Axis (HPTA) suppression and closure to the extent where they might require far more extended periods to ensure full recovery than most. This, like anything else, is a spectrum whereby there are the very ‘lucky’ individuals that recover very quickly and easily on one end of the spectrum, and the ‘unlucky’ individuals that have extreme difficulty recovering during post cycle therapy. In between the two extremes is the average. Once again, this is due to the individual’s genetic programming as to how the Hypothalamic Pituitary Testicular Axis (HPTA) will respond and attempt to maintain homeostasis.

Type of Testosterone Therapy(s) used:

All testosterone therapy exhibit suppression or shutdown of the Hypothalamic Pituitary Testicular Axis (HPTA) through the mechanisms of the negative feedback loop, and there are no exceptions to this. Various testosterone therapy are known as being mildly suppressive, while others are identified as being profoundly suppressive. This is all reliant on multiple different reasons, many of which will not be discussed here. In any case, no matter how mild or severe an testosterone therapy exerts Hypothalamic Pituitary Testicular Axis (HPTA) suppression, all testosterone therapy when utilized for typical cycle lengths of weeks at a time will eventually cause the Hypothalamic Pituitary Testicular Axis (HPTA) to shut down, or at the very least severely suppress its hormonal signal processes.

Length of the cycle degree of testicular desensitization:

This is perhaps the most important and most influential factor. As the range of testosterone therapy use continues, the majority of the Leydig cells of the testes remain dormant and inactive, and the longer these interstitial cells stay dormant and idle, the higher the difficulty is essentially getting these cells to respond to the stimulus of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) once again. It has been discovered in studies that the issue of recovery of the Leydig cells following testosterone therapy use is not due to a lack of Luteinizing Hormone (LH), but due instead to the desensitization of the Leydig cells to (LH). In one study in which exogenous Testosterone was administered to male test subjects for 21 weeks, Luteinizing Hormone (LH) levels were suppressed shortly after beginning administration. However, at the end of the 21 weeks, Luteinizing Hormone (LH) levels were observed to rise within three weeks once the exogenous Testosterone administration stopped, but Testosterone levels did not arise until many weeks later in most of the test subjects.

Recovery During The Post Cycle Therapy (PCT).

To stimulating hormonal recovery during post cycle therapy, it is essential for individuals to understand that the use of any medication except for a single select one or two is inadequate for hormonal recovery during post cycle therapy (PCT). Ideally, all post cycle therapy programs should be a multi-component post cycle therapy (PCT) program that includes several different compounds that work in tandem with one another to provide the most effective and fastest possible Hypothalamic Pituitary Testicular Axis (HPTA) recovery following an testosterone therapy.

The three categories of compounds are in order of importance:

  • SERMs (Selective Estrogen Receptor Modulators)
  • Aromatase Inhibitors
  • HCG (Human Chorionic Gonadotropin)

SERMs:

Classes of drugs in the SERM category include: Nolvadex (Tamoxifen Citrate), Clomid (Clomiphene Citrate), Raloxifene, and Fareston (Toremifene Citrate). The nature of a SERM is that it exhibits mixed Estrogen agonist and Estrogen antagonist effects on the body. This means that although a SERM might block the effect of Estrogen at the cellular level in specific tissues, it can enhance Estrogenic impacts in other areas of the body. These can be positive effects as well as adverse effects. Nolvadex, for example, exhibits Estrogenic agonistic effects in the liver, which is a positive effect, as its effects here result in a positive change in cholesterol profiles (something desired by many). All SERMs to varying degrees serve to act as an Estrogen antagonist in this area, working to mitigate Estrogen’s effects on breast tissue, reducing or blocking the side effect of gynecomastia. Regarding the impact of SERMs on endogenous Testosterone stimulation, they serve to act as an Estrogen antagonist at the pituitary gland, triggering the release of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) as a result. Elevated levels of Estrogen in men can and does suppress the output of endogenous Testosterone via the negative feedback loop, leading to hypogonadism. SERMs for this purpose are an essential addition to any post cycle therapy (PCT) protocol and are not to be excluded under any circumstance. Regardless of this, however, the sole focus should not be on SERMs.

Aromatase Inhibitors:

These are compounds such as Aromasin (Exemestane), Arimidex (Anastrozole), and Letrozole (Femara). Rather than block the activity of Estrogen at the cellular level in different tissues, aromatase inhibitors (AIs) serve to lower total circulating Estrogen levels in the body by way of inhibiting the aromatase enzyme, which is the enzyme responsible for the conversion of androgens into Estrogen. The transformation of androgens into Estrogen results in excess Estrogen levels, which, as explained earlier in this article, will trigger the negative feedback loop leading to suppression of Testosterone production. By way of lowering total circulating blood plasma Estrogen levels, AIs will positively engage the negative feedback loop and result in the release of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) for the manufacture and secretion of more Testosterone. This is mainly due to the hypothalamus realizing that circulating. Estrogen levels are too low and will attempt to increase circulating levels of Testosterone for a portion of the Testosterone secreted to be able to become aromatized into Estrogen to restore the hormonal balance. The other importance of aromatase inhibitors is the ability to mitigate the Estrogenic effects of human chorionic gonadotropin (HCG), which will be explained shortly. It is important to note, however, that the majority of aromatase inhibitors do not comply very well with SERMs such as Nolvadex, and those particular choices should be made in regards as to which AI is used during post cycle therapy (PCT).

HCG:

Human Chorionic Gonadotropin is, for the most part, synthetic Luteinizing Hormone (LH). It is a protein hormone manufactured in high amounts by pregnant females that contains a protein subunit that is 100% identical to Luteinizing Hormone (LH), and therefore when administered to men, it will mimic the action of Luteinizing Hormone (LH) in target tissues, such as the testes. What results is an increase in Testosterone production via stimulation of the Leydig cells by human chorionic gonadotropin (HCG). Human chorionic gonadotropin (HCG) should never be utilized alone, as its nature as a gonadotropin will itself trigger a negative feedback loop whereby once human chorionic gonadotropin (HCG) is used, the pituitary gland will halt output of Luteinizing Hormone (LH) until human chorionic gonadotropin (HCG) use has discontinued. Therefore, human chorionic gonadotropin (HCG) must be utilized with a SERM and especially an aromatase inhibitor, as human chorionic gonadotropin (HCG) has demonstrated to increase aromatase activity in the testes, resulting in rising Estrogen levels.

Putting Them Together:

The reader may be wondering which compounds to select of the three categories listed, and how to use them properly. The answer lies in understanding the properties of each and, in interpreting these properties, how to use them efficiently and appropriately.

Human chorionic gonadotropin (HCG):

The first item to be examined will be human chorionic gonadotropin (HCG). The majority of testosterone therapy users from the 1960s – mid-1980s did not even utilize any compounds for hormonal recovery, and the term post cycle therapy (PCT) did not even exist at that time. When the use of human chorionic gonadotropin (HCG) became increasingly popular (circa 1980), it was the only compound utilized. Since then, the medical and scientific understanding of such things has increased exponentially, and there should be no reason for any informed and adequately educated individual to utilize human chorionic gonadotropin (HCG) on its own for post cycle therapy (PCT). When used in conjunction with one of the other two categories of compounds (an AI and a SERM), the dynamics change considerably.

It has been mentioned already that much of the difficulty in recovering the Hypothalamic Pituitary Testicular Axis (HPTA) following an testosterone therapy is the result of Leydig cell desensitization. Human chorionic gonadotropin (HCG) is necessarily an analog of Luteinizing Hormone (LH), and the testes after a prolonged testosterone therapy would be as equally desensitized to human chorionic gonadotropin (HCG) as they are to Luteinizing Hormone (LH). The human body, however, produces Luteinizing Hormone (LH) amounts on its own that is far too inefficient for proper and rapid Testosterone production. The body’s natural increase of Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH), evidenced by the study referenced earlier in which it was not until three weeks when Luteinizing Hormone (LH) levels only began to reach the standard physiological measurements following the cessation of Testosterone. Therefore, the body’s natural Luteinizing Hormone (LH) production does not provide a high enough dose for stimulation, nor an immediate stimulus to the tests required for the initial increase in Testosterone needed during the post cycle therapy weeks.

Human chorionic gonadotropin (HCG), utilized in a specific manner during the first 1 – 2 weeks of post cycle therapy (PCT) at a dose of 100-1,500IU every 2 days, is what allows the individual to provide the testes with a high dose to provide them with a ‘shock’ effect, and sustain this shock effect on the Leydig cells of the testes for a sustained period of the first 1 – 2 weeks of post cycle therapy. Studies have demonstrated the incredible effectiveness of human chorionic gonadotropin (HCG) for this purpose, it been suggested that human chorionic gonadotropin (HCG) therapy is utilized to treat low testosterone and hypogonadism. Following this line of thought, the other two compounds (the SERM and the AI) are to be used as supportive compounds for human chorionic gonadotropin (HCG) use in this 1 – 2 week period, and after human chorionic gonadotropin (HCG) is discontinued early on in post cycle therapy (PCT), only the SERM is to be used in order to carry along the hormonal recovery process.

In spite of the good news in regards to the ability for human chorionic gonadotropin (HCG) to assist in hormonal recovery, there are still two remaining issues to be addressed:

  • The fact that human chorionic gonadotropin (HCG) causes increased production of aromatase, leading to increased Estrogen levels.
  • Following the discontinuation of human chorionic gonadotropin (HCG), the body is left with very little endogenous Luteinizing Hormone (LH), and Follicle Stimulating Hormone (FSH) production due to the exogenous administration of human chorionic gonadotropin (HCG).

Aromatase Inhibitors:

Aromasin (Exemestane) Above All Else The first of the two remaining issues to be addressed will be the fact that human chorionic gonadotropin (HCG) will trigger increases in testicular aromatase expression, and result in Estrogen increases in the body. It should also be noted that it will cause an increase in testicular progesterone levels. Estrogen rising is, of course, undesirable during post cycle therapy (PCT), as it has already been explained that Estrogen will trigger suppression of endogenous Testosterone production, and there is no doubt that any individual wishes to encounter Estrogenic side effects during post cycle therapy (PCT) either.

Therefore, the option here is to include an aromatase inhibitor. However, there exists a big problem in regards to the other two of the three major aromatase inhibitors (Arimidex and Letrozole). The issue is the fact that in a post cycle therapy (PCT) program that includes the use of SERMs such as Nolvadex and Clomid, which are known as essential components to a post cycle therapy (PCT) program, Arimidex and Letrozole have direct negative interactions with Nolvadex. The problem here is that Arimidex (or Letrozole) and Nolvadex both directly counteract one another. One study has demonstrated that when Arimidex is utilized with Nolvadex, Nolvadex will decrease the blood plasma concentration of Arimidex (as well as Letrozole, another commonly used aromatase inhibitor). The conclusion here is that the use of Arimidex or Letrozole with Nolvadex together is a terrible idea and may work together in a post cycle therapy (PCT) protocol. Aromasin completely circumvents this problem, as it has been demonstrated to have no interactions what so ever with Nolvadex, unlike the other two aromatase above inhibitors. In one study, Aromasin displayed no such reduced effectiveness or any reduced blood plasma levels when utilized with Nolvadex.

The other benefit of selecting Aromasin over all other AIs is the fact that Aromasin has demonstrated in several studies to impact cholesterol profiles in a negative manner far less than other aromatase inhibitors have, wherein one particular review on cancer patients, 24 weeks of Aromasin (Exemestane) administration held no impact on cholesterol profiles. Some other studies have also demonstrated a nil effect on cholesterol profiles from the use of Aromasin. Although there have also been some studies that have shown a negative impact on cholesterol profiles resultant from Aromasin use, it is evident that there is not as a significant or as a negatively impacting effect from Aromasin on cholesterol as other aromatase inhibitor.

Finally, in addition to these benefits from Aromasin, it is evident that Aromasin holds the ability to increase Testosterone levels in males as demonstrated by studies. For example, one particularly notable study selected 12 healthy young male test subjects, and were administered random Aromasin doses of 25mg and 50mg for a 10 day period, and not only was Estrogen suppressed by a significant amount (38%), but Testosterone levels in the test subjects were observed to have increased by an incredible 60%.

Following these details, Aromasin would be the best possible aromatase inhibitor of choice to combat the increased aromatase activity caused by human chorionic gonadotropin (HCG). Therefore, Aromasin would then be utilized at a full 25mg daily dose, and only while human chorionic gonadotropin (HCG) is used. Once human chorionic gonadotropin (HCG) is discontinued, Aromasin too should be halted.

The only following issue to cover now is that of stimulating and maintaining proper endogenous Luteinizing Hormone (LH) release to carry recovery along until the body can become self-sufficient once again.

Nolvadex and Clomid: 

The question is often asked among the testosterone therapy using community: Clomid or Nolvadex? Which one for post cycle therapy (PCT)?

First of all, the best possible addition to human chorionic gonadotropin (HCG) in a post cycle therapy (PCT) protocol is Nolvadex (Tamoxifen Citrate), as studies have demonstrated that human chorionic gonadotropin (HCG) and Nolvadex utilized together have exhibited a remarkable synergistic effect in terms of stimulating endogenous Testosterone production and that Nolvadex will actually work to block the desensitization effect on the Leydig cells of the testes caused by high doses of human chorionic gonadotropin (HCG). This is very important because just as too little Luteinizing Hormone (LH) secretion for extended periods can cause desensitization to gonadotropins, too much gonadotropin stimulation (in the form of human chorionic gonadotropin (HCG) or otherwise) will likewise create a desensitization effect.

Secondly, Nolvadex on an mg for mg basis is far more effective than Clomid in stimulating endogenous Testosterone production, as well as being a more cost-effective choice than Clomid itself. Studies have demonstrated that 150mg of Clomid (Clomiphene Citrate) administered daily raised endogenous Testosterone levels of 10 healthy males by approximately 150%, while incidentally, 20mg of Nolvadex (Tamoxifen Citrate) daily raised endogenous Testosterone levels by the same amount. It is very evident here that Clomid is very useful for this purpose, but Nolvadex seems to be a more cost-effective choice seeing as though it is more effective than Clomid when compared mg for mg. The benefits of Nolvadex over Clomid do not end there – Clomid, although it does exhibit Estrogen antagonist effects at the pituitary gland as Nolvadex does, actually shows Estrogen agonist effects there too. What this means is that Clomid will work in varying degrees as an Estrogen at the pituitary gland, triggering the negative feedback loop and reducing the output of Testosterone stimulating gonadotropins Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH). This is a severe problem during post cycle therapy, which is a period in which individuals are trying to recover their Hypothalamic Pituitary Testicular Axis (HPTA) function rather than halt it even further. Ideally, one would want a SERM that exhibits almost 100% Estrogen antagonistic effects on the pituitary gland, and Nolvadex is the perfect choice for this.

When it comes to the dosing aspect of Nolvadex, The standard dose for post cycle therapy (PCT) and for stimulating the release of GnRH (Gonadotropin Releasing Hormone), Luteinizing Hormone (LH), Follicle Stimulating Hormone (FSH), and ultimately Testosterone is that of a single Nolvadex dose of 20 – 40mg daily. In all studies involving Nolvadex doses used to stimulate endogenous Testosterone production, only 20 – 40mg daily of Nolvadex was utilized, and it has been shown that doubling the dose to 40mg or any higher will not produce any significant difference in endogenous Testosterone secretion. The only reason why many elects to utilize 40mg daily of Nolvadex for the first 1-2 weeks of a post cycle therapy (PCT) program is to achieve optimal peak blood plasma levels quicker to ensure Hypothalamic Pituitary Testicular Axis (HPTA) recovery faster.

The ideal post cycle therapy protocol for 4 – 6 weeks Total post cycle therapy (PCT) time (depending on the recovery ability of the individual):

01) Weeks 1 – 2:

  • human chorionic gonadotropin at 1000iu/E2D.
  • Aromasin (Exemestane) at 25mg/day.
  • Nolvadex (Tamoxifen Citrate) at 40mg/day.

02) Weeks 2 – 6:

  • Nolvadex or (Tamoxifen Citrate).

Additional and Optional, Vitamins, Supplements, Compounds to Aid During post cycle therapy (PCT), Aside from the principal components discussed, various other parts are mostly optional, but still very useful for hormonal recovery of the Hypothalamic Pituitary Testicular Axis (HPTA) during the post cycle therapy weeks.

Vitamin B12 Health Benefits, There has been a lot of controversy over whether or not there is indeed a benefit from taking B12 supplements. Some doctors suggest that as long as a person is not vegan (though probiotics in the gut can produce some B12), they are probably getting sufficient B12 from the basic foods they are ingesting.

vitamin b12

Let us reference some studies:

There is documented research from the Framingham Study suggesting that 40% of all people are deficient in B12. The American Journal of Clinical Nutrition researched in 2009, and they published a study suggesting close to 6% of U.S. and U.K. residents over the age of 60 are B12 deficient. Another 20% were referenced as “marginal status.”

Vitamin B12 Health Benefits has the most multifaceted and prevalent chemical structure of all vitamins. One area where it differentiates from other vitamins is in the fact that it’s the only vitamin that contains a metal commonly referred to as Cobalamin, which is also a universal term for all the various compounds that may have some B12 properties in it.

B12 can improve energy by aiding in thyroid function and cellular methylation, That being said, B12 is not only useful in supporting healthy energy levels. It is unequivocally essential to life and whole existence. People deficient in B12 will suffer from serious health issues if the problem is not addressed.

What role B12 plays in the following human biological processes:

  • Nerve and brain regeneration
  • Adrenal gland support
  • Male and female reproductive health
  • Nutrient absorption
  • Red blood cell formation
  • Cellular energy
  • Memory recall
  • DNA synthesis

Here are some of the risks associated depleted B12 levels are:

  • Pernicious anemia
  • Migraine headaches
  • Macular degeneration
  • Tinnitus
  • Fatigue (adrenal fatigue and CFS)
  • Multiple sclerosis
  • Memory loss
  • Neuropathy
  • Anemia
  • Asthma
  • Shingles
  • Kidney disease
  • Depression

August 10, 2018 by Joseph Fermin 0 Comments

Nutraceutical and Weight Loss Therapy 5 (6)

Nutraceutical and Weight Loss

Weight Loss therapy

There are now millions upon millions of options for weight loss therapy and nutraceuticals available on the market today. Many fads that, for the most part, do actually work, but the end result for 99% of the participants is always the same; gaining back more weight loss therapy they actually started off with.

The one thing that these diet programs have all wrong is the notion that the “same” therapy program will work for every single person that tries it. That is propaganda. Whoever tries to convince you that if something worked for one person, then it must work for you as well, is lying to you and does not have your best interest in mind. Another very important, yet logical characteristic of “being thin” is the actual need to change your entire lifestyle… that is literally the only way to weight loss therapy and keep it off. If you go back to what you were doing before, you will just end up the way you looked and felt before.

Now that that’s out of the way, let’s talk about options available to help you on your path to the new you. Options that may even help you to accelerate the process a bit. For the core group, the whole point of founding AAI Rejuvenation Clinic was to establish an organization based on the premise that patient care and focus, really putting the patients first, is of utmost importance and the kind of focal structure missing in most medical establishments today. Our logical determination is that if we actually focus on helping patients and honestly answer queries related to their health and wellness goals, our patients will be loyal to our process and to our establishment; happy to be a part of the AAI Rejuvenation Clinic family and satisfied with our level of service, respect and, overall, their final goals and end results.

All Weight Loss Therapy are NOT Created Equal!

Medicinal therapies and protocols cannot be a one-size-fits-all structure. Each individual patient’s specific physiological characteristics, their daily routine, as well as their actual goals and aspirations must be taken into consideration when drawing up and organizing a plan of attack to drop those unwanted pounds and inches.

For the most part, the nutritional diagnosis will be the focus of your protocol assembling. Additionally, hormone treatments are very commonly incorporated into most diet regimens to help patients needing the additional assistance in weight loss due to physiological apprehensions.

Though Gonadotropin is one of the options available at AAI Rejuvenation Clinic as a weight loss therapy program, the reality is that it is not one of the healthiest weight loss options available out there and we do try to stray from incorporating it into our agendas. Needless to say, it is a program that helps some participants lose their desired weight and it can be effective if very properly administered and unmistakably followed. What we do different, in cases where interested patients qualify for these another hormone related weight loss options, such as the Gonadotropin hormone replacement program, is we integrate an array of vitamins, probiotics, amino acids and other such supplementing pharmaceuticals in an effort to cater to the immune system, which is one of the strongest downfalls of participating in such a protocol, which requires such a minimal amount of caloric consumption daily.

Why AAI Rejuvenation Clinic is Different.

At AAI Rejuvenation Clinic, we do what is best for your body in terms of helping you reach your health and wellness goals. We’ve learned that many times when a patient is having problems reaching their weight goals, the association has to do with a bad combination of food choices for that individual specifically. The way the body processes food is very different from one person to another, so understanding a person’s eating habits in conjunction with proper and adequate blood tests can really help zero in on a program, and the right supplementing nutraceuticals, to help eliminate whatever toxins may be preventing them from losing weight or, whatever hormone imbalance may be slowing down their weight loss process. Whatever metabolic issue may be obstructing a person’s ability to reach their weight loss goals can be addressed once it’s properly diagnosed.

July 31, 2018 by Joseph Fermin 0 Comments

The 6 Effects of Testosterone in Your Body 4.8 (6)

The 6 Effects of Testosterone in Your Body

6 Effects of Testosterone in Your Body, However, the body changes as we age and loses some of its regenerative capacity. A fundamental part of this decline roots from a slow-down in our hormonal secretions. Simply put, even young people with glandular issues who do not produce enough testosterone and/or HGH experience side effects and maladies that are usually only associated with much older individuals. Conversely, older patients with higher hormone levels, be it naturally or via hormone replacement therapy, have health benefits normally associated with younger individuals.

Also, Women’s Can Be at Risk for Low Testosterone:

The ovaries are responsible for producing both testosterone and estrogen, as the ovaries age, they produce less estrogen and testosterone. As women enter peri-menopause and pre-menopause, testosterone will be diminished by the age of 30’s, and once a women reach full menopause, it’s common for them to produce 75% less testosterone than they did in the there 21’s. Every woman becomes at risk of low testosterone as she ages, and women who go through hysterectomy or oophorectomy have an even higher chance of dealing or having low testosterone in women levels.

6 Effects of Testosterone in Your Body.

  • Changes the Brain:

Research has shown that women have a significant advantage when it comes to a few things, Like memory and language, while men tend to have stronger spatial skills (though this too has been disputed). But due to ethical restrictions, no study had been able to track the direct effect that testosterone exposure has on the brain—until now.

The hormone also plays a role in your state of mind, including how well your brain works. That’s why low testosterone has linked to symptoms such as mood swings, increased stress, and depression. … “Cells in the brain have testosterone receptors, and low testosterone in those receptors are significantly affected mental health.”

  • Muscle and Strength:

Recent studies have shown that men as they age, they lose more muscle mass than women of the same period, and has led researchers to believe that the loss of muscle strength and has it has a direct link to the predominantly male decline hormone, like testosterone. Testosterone helps to build muscle mass and strength in young men.

Testosterone supplementation in men increases fat-free mass, but whether measures of muscle performance, such as maximal voluntary strength, power, fatigability, or specific tension, are improved has not been determined. … A seated leg press exercise defined maximal voluntary muscle strength and fatigability.

  • Bone Density and Osteoporosis:

As men age, their serum testosterone concentrations decrease, as do their bone densities. Because bone density is also low in hypogonadal men, we hypothesized that increasing the serum testosterone concentrations of men over 65 yr to those found in young men would raise their bone densities.

Testosterone increases levels of growth hormone. That makes the exercise more likely to build muscle. Testosterone increases bone density and tells the bone marrow to manufacture red blood cells. … Dropping the levels of testosterone can cause an increase in body fat.

  • Sex Drive and Libido:

Some men maintain sexual desire at relatively to low testosterone level. For other men, libido may lag even with normal testosterone levels and low testosterone is one of the possible causes of low desire and sex drive, however. If testosterone has lowered far enough, virtually all men will experience some decline in sex drive.

Growth hormone (GH) has also been used to reverse or correct erectile dysfunction and other sexually impeding disorders and the primarily by improving circulation and blood flow, which results in a firmer, more sustainable erection.

  • Bone Marrow and Red Blood Cells:

Erythropoiesis affects the production of new red blood cells. One is accomplished primarily in the bone marrow, the red blood cell factory. Erythropoiesis is stimulated mainly by erythropoietin (hence, the clever naming of that hormone!). … Androgens, including testosterone, are another type of hormone.

Testosterone and other androgens have an erythropoietic stimulating effect that can cause polycythemia, which manifests as an increase in hemoglobin, hematocrit, or red blood cell count. … If hematocrit elevated before starting testosterone, the cause should determine before beginning androgen therapy

  • Skin and Collagen:

A man or women with shrinking levels of testosterone actually may lose some body hair. Testosterone replacement therapy comes with a few potential side effects, including acne and breast enlargement. Testosterone patches may cause minor skin irritation.

In fact, research now suggests that bioidentical hormone replacement therapy (or BHRT) can actually reverse many of the signs of aging and that you may have come to accept – weight gain, low libido, reduced muscle mass – and can even help to prevent conditions such as osteoporosis, heart disease, and dementia.

 

(more…)

June 25, 2018 by Joseph Fermin 0 Comments

Body Mass Index (BMI) Measure of Body Fat 5 (4)

Body Mass Index (BMI) and How it Measure the Body Fat

Body Mass Index (BMI) is based on your high and weight about your height and applied to most adult men and women aged 20 and over. For children aged 2 and above, BMI percentile is the best assessment of body fat.

BMI does not measure body fat directly. However, research indicates that BMI correlates to direct measures of body fat such as underwater weighing and dual-energy X-ray absorptiometry (DXA), and is considered an inexpensive and easy-to-perform alternative for these.

FACTS ON BMI

Here are some critical points about BMI:

  • Body mass index (BMI) is a measure of relative size based on the mass and height of an individual.
  • The Quetelet Index devised by Adolphe Quetelet, a Belgian mathematician, astronomer, and statistician, in 1832.
  • It was later termed “body mass index” in 1972 by Ancel Keys.
  • BMI is a simple, inexpensive and noninvasive surrogate measure of body fat.
  • Factors such as age, sex, ethnicity, and muscle mass do not account in BMI chart.

For adults over 20 years old and older, BMI can be interpreted by using standard or not updated there weight status, and categories that are the same for all ages, and for both women and men.

For children, kids and adolescents between 2-20 years old, BMI interpreted relevant categories to a child’s age and sex. BMI is reasonable throughout the years as an indicator of body fat categories for both adults and children. Because BMI does not measure categories of body fat directly, it should not use as a diagnostic tool.

BMI should be used only to measure to track weight status in populations and also as a screening categories tool to identify potential weight problems in individuals. Body fat measures, such as skinfold thicknesses, bioelectrical impedance, underwater weighing and dual energy X-ray absorption, maybe more accurate than BMI.

WHAT IS BODY MASS INDEX (BMI)?

BMI use as a screening tool to indicate whether a person is an underweight, healthy weight for their height, overweight or obese. If a person BMI is out of the excellent measure by the BMI range, their health risks may increase.

obesity-in-the-world

BMI is age-independent, the same for both women and men. But the BMI may not be the same degree of fat in different populations because due to a diverse body and other proportion. The health risks associated with high BMI can be confusing, the interpretation of BMI risk may differ for different populations.

There are three ways to check your BMI:

  • Use our BMI calculators
  • First, use the BMI chart, find your height in the left-hand column. Then, follow it over until you see your weight along the top bar.
  • The number in the center box between the two is your BMI.

THE BENEFITS OF MAINTAINING A HEALTHY WEIGHT

  • Fewer joint and muscle pains
  • The ability to join in more activities and Increased energy
  • Improved bodily fluids and blood pressure
  • The Reduced burden of the heart and circulatory system
  • Improved sleep patterns

Reductions in blood glucose, the risk of developing type 2 diabetes and triglycerides:

  • Excess of weight increases how hard the heart has to work to pump blood, it also gives high blood pressure, blood cholesterol and triglyceride levels and lowers HDL (good) cholesterol levels.
  • Excess of weight can make help likely to develop any diabetes.
  • Lifestyle changes that help maintain a 3-5% weight loss and likely to result in clinically meaningful improvements in blood glucose, triglycerides, and risk of developing type 2 diabetes.
  • Greater weight loss can and also help reduce blood pressure and improve blood cholesterol.

THE BODY MASS INDEX MEASUREMENTS?

Researchers point out that the (BMI), based on the height and weight, is not an accurate measure of body fat because it does not account for critical factors that contribute to health or mortality, such as fat distribution, the proportion of muscle to fat, and the sex and racial differences in body. However, it used as a screening tool to identify potential weight problems in adults.

If a person has a high BMI, But yet to determine if this excess weight is a health risk, call your doctor, or a healthcare provider would need to complete further assessments such as:

  • Skinfold thickness measurements
  • Evaluations of diet
  • Physical activity
  • Family history
  • Other appropriate health screenings

HOW TO CALCULATE BMI FOR ADULTS

Belgium Statistician Adolphe Quetelet developed the BMI formula approximately 150 years ago.

THE CALCULATION OF BMI

BMI based on the following formulas:

  • Metric BMI Formula. Weight (kg) / Height (m)2

With the metric system, the formula for BMI is weight in kilograms divided by height in meters squared (kg/m2). Since height commonly measured in centimeters, divide height in centimeters by 100 to obtain height in meters.

  • Imperial BMI Formula. (Weight (lbs.) * 703) / Height (inches)2

December 20, 2017 by Joseph Fermin 0 Comments

How Testosterone Affects Fat Loss: Real Science of Low-T 5 (1)

How Testosterone Affects Fat Loss: Real Science of Low-T

Testosterone Affects Fat Loss, As men age, their testosterone levels decline. This increase in SHBG (sex-hormone binding globulin), which binds to testosterone and thus reduces the amount of free testosterone in the body. Low testosterone levels, obesity, and many other health problems, including fatigue, memory problems and decreased muscle and bone mass. So what is the impact of testosterone on the body and how Testosterone Affects Fat Loss?

Testosterone and energy – low levels of testosterone can cause low energy levels. Regaining natural testosterone levels can increase strength and lead to weight loss. Studies have found that testosterone supplementation reduces total body fat percentages. Obesity is related to low testosterone levels in men. Just as testosterone therapy is associated with decreased obesity, losing weight is associated with increased testosterone levels.

An article in 2014 in the journal of Current Opinion in Endocrinology, Diabetes, and Obesity looked at the existing data regarding testosterone therapy in overweight and obese men with testosterone deficiency (hypogonadism).

  • Many weight loss supplements have dangerous side effects or are ineffective
  • Looked at weight loss, BMI, waist circumference and body composition
  • Evaluated long-term testosterone therapy
  • About 40% of obese nondiabetic men and 50% of overweight diabetic men over 45 have low levels of free testosterone
  • Many studies have found that testosterone therapy in overweight men helps increased lean body mass, weight loss, decreased BMI and reduced waist circumference.
  • Weight loss is significant and sustained with long-term testosterone therapy
  • Testosterone plays a part in protein, fat and carbohydrate metabolism.
  • Testosterone is involved in mitochondrial function and thus energy production and utilization
  • This interference with energy production is likely why those with low testosterone levels experience a lack of energy
  • Testosterone therapy has been found to increase lipid oxidation and normalize glucose use in the body. It was found to increase strength and motivation.

Weight gain associated with an 88% increase in body fat and a 12% increase in lean body mass (LBM) and weight loss with a 72% decrease in body fat and a 28% decrease in LBM.

  • With testosterone therapy, LBM has been shown to increase.
  • Increased LBM means increased energy expenditure at rest
  • Testosterone therapy with improved cardiometabolic function

Testosterone deficiency decrease in energy metabolism from fat and an increase in energy metabolism from glucose.

  • Testosterone therapy has been shown to increase lipid metabolism

Safety? Safety is the primary concern with testosterone replacement therapy. Possible risks discussed in the literature include:

  • Increased risk of prostate and breast cancer
  • Liver toxicity and tumors
  • Sleep apnea exacerbation
  • Testicular infertility and atrophy

Talking with your doctor before starting testosterone injections therapy is necessary.

Summary: If you are a male struggling to lose weight, testosterone therapy is a promising treatment to help with not only weight loss but for body composition, energy levels and overall health.

November 13, 2017 by Joseph Fermin 0 Comments

An Inside Look at Testosterone Injections Therapy 5 (1)

Inside Look Testosterone Injections Therapy

testosterone injections therapyTESTOSTERONE INJECTIONS THERAPY

Testosterone is an essential factor in males, that does more for men than just promote sex drive. Low Testosterone levels decline as a part of healthy aging and are the cause of several physiological changes. Low Testosterone level symptoms include;

1) Reduced Motivation
2) Erectile Dysfunction
3) Loss of Libido or sex drive
4) Fatigue and low energy
5) Increased Cholesterol Levels
6) Memory and Concentration
7) Decreased Muscle Mass
8) Thyroid Dysfunction and more

Since these symptoms are common in low testosterone, physicians will often include hormone levels as part of routine blood work. Normal levels of testosterone are between 300 and 1,000 ng/dL. If a blood test shows that your levels are far below the norm, your doctor may suggest testosterone injections. The treatment is called TRT.

SYMPTOMS OF LOW TESTOSTERONE

Most men naturally start losing testosterone when they hit their 30’s. (low T). Common symptoms of low Testosterone include:

1) Erectile dysfunction (ED)
2) Changes in sex drive
3) Decreased sperm count
4) Depression or anxiety
5) Weight gain
6) Hot flashes

TESTOSTERONE AND DIAGNOSIS

Many men may want to diagnose themselves with a testosterone kit. The problem with self-diagnosis is that many of the symptoms of low Testosterone are healthy parts of aging. So using it for diagnosis isn’t reliable. Our doctor may order testosterone blood test. It is the only way to find out if you have low testosterone.

To get a perfect reading our doctor will take a look at your health history, physical exam and blood test to measure your testosterone levels. You’ll also likely have a test that measures your red blood cell count.

POTENTIAL BENEFITS OF TESTOSTERONE INJECTIONS THERAPY 

The purpose of Testosterone Injections Therapy is to help regulate hormone levels and to help address problems related to low Testosterone. For men with low Testosterone, the benefits of these injections can include:

1)  Motivation and Memory Loss
2)  Sex Drive & Desire
3)  Depression and Energy
4)  Cholesterol and Osteoporosis
5)  Erectile Dysfunction
6)  Muscle Mass and Better Sleep
7)  Wounds healing & Illness
8)  Thyroid Dysfunction and more

TESTOSTERONE CAN HELP WITH FAT AND MUSCLE CHANGES

Men have less body fat than women; This is partly related to testosterone, which regulates the fat in the body and muscle maintenance in your body. You’ll likely also notice an increase in body fat, especially around your midsection.

Your hormones also help regulate muscle growth. So, with low Testosterone, you may feel like you’re losing muscle size or strength.

Testosterone shots regulate fat distribution, but you shouldn’t expect significant weight loss changes from hormone therapy alone, without exercise. As for maintenance of muscle, testosterone therapy has been found to improve increase muscle mass, but not strength.

TESTOSTERONE INJECTIONS THERAPY AND SPERM COUNT

Low sperm count in men is a common side effect of low Testosterone. This problem can make it difficult to get your partner pregnant.

TESTOSTERONE INJECTIONS THERAPY AND THE BOTTOM LINE

Testosterone injections therapy can only be helpful If you just have low Testosterone. If you’re wondering if testosterone is a right choice for you, ask your doctor. They can test you for low Testosterone. Ask your doctors, or Give us a call, if testosterone injections therapy would be a good choice for you.

If you don’t end up having low Testosterone but still feel like your hormone levels might be off, keep in mind that proper Food, Regular exercise, could help you increase testosterone naturally and make you feel better. If that doesn’t help, be sure you contact us for help.

August 25, 2016 by admin 3 Comments

Anavar: The Good, The Bad and The Ugly 5 (1)

Pros and Cons of Anavar

Anavar has been around since 1964. It was bought out by Pfizer in 2003. Since then, it has seen a popular reemergence and has been termed the “safest” steroid available. Unfortunately, this is dangerous and brings confusion to the masses whom misleadingly supplement with this compound.

Anavar

We have been receiving many requests for Anavar from patients that have shopped around with other local clinics. Sadly, clinics that, despite the consequential ramifications, opt to sell this controlled substance to their patients. Moreover, they do it under the guise of providing medical treatment, when in fact, this is one of the reasons it was once illegal: there is no real medicinal purpose for Anavar.

  • Anavar is an anabolic steroid.
  • Anavar is not a bio-identical hormone, as is testosterone.
  • Anavar has very little androgenic (testosterone Injections) properties. This is the reason it does not aromatize. Only androgens aromatize.
  • Initially, Anavar was produced to assist patients with lipodystrophy (excessive muscle loss, usually as a result of AIDS).

The Good, The Bad, and The Ugly.
First the Good

  • Because there is no conversion to estrogen with Anavar, blockers nor inhibitors are needed for its use. Estrogen conversion manifest most of the visible side effects men fear from a testosterone therapy program: mood swings, libido loss, water retention, weight gain, etc.
  • Anavar increases anabolism significantly. This allows the muscle to absorb a lot more protein. Being in anabolic state intensifies the muscle building process. Strong, dense muscles develop and are longer lasting than when developed on other steroids.
  • Because Anavar has almost no androgenic properties, it does not cause a swift shutdown of HPTA (the process that invites endogenous low testosterone production)
  • Muscles developed with the aid of Anavar administration are sharper and more cut.
  • Studies have shown Anavar to diminish visceral (stomach area) fat with only moderate exercise. Most impressively, even after discontinuing usage, the subjects kept off the fat. Of course, they were still exercising and minding their caloric intake.
  • Anavar can lubricate joints and assist with joint related pain.

The Bad

  • Want to know the truth behind why Anavar is considered the “safest” or “most mild” anabolic steroid available? In its pharmaceutical form, Anavar comes in 2.5 mg tablets. At that therapeutic dose, even a child could take it without systemic repercussions. There is basically no negative impact on the liver, even if taken on a daily basis.
  • To build significant muscle on Anavar alone, at least 50 mg a day would be needed. At these quantities, arduous taxing of the liver is intense and inevitable.
  • Anavar is a seventeen alpha alkylated. This means it is structured to prevent a breakdown in the liver. This makes the effects of the drug much greater but puts an unimaginably damaging strain on the liver.
  • The required dosage for effective muscle “building” is what makes legitimate medical claims of Anavar’s “mildness” a myth.
  • In a nutshell: With Anavar, what makes it safe, makes it not very effective, and once it becomes effective, it is not very safe.

The Ugly

  • 2.5 mg is the pharmaceutical dosage this brand medication comes in. Patients should be wary of 20 mg to 50 mg tablets. Those are only made through UG laboratories. Patients should require their medication come labeled with their name, the prescribing physician’s name and the providing Laboratories information, including DEA number. This info should be followed up with some quick research on the involved laboratory.
  • Unless you are suffering from acute muscle wasting, there is no medicinal purpose for taking Anavar.
  • Anavar is actually pretty amazing stuff “if you are a bodybuilder willing to administer illegal substances.” It does have the ability to increase strength significantly in a short period of time. However, at the dosages required for effective muscle building, the liver finds itself under constant attack. This program can only be used as a kick-start program.
  • Patients prescribed Anavar under the guise of it being a health supplement should consider very carefully the integrity of the organization they are working with.
  • A “real” Anavar program should not surpass 4 to 6 weeks. Anything beyond that, especially at dosages beyond the 20mg mark, will have impacting effects on the liver.
Real Testosterone Therapy

anavarIn conclusion, Anavar, if abused, is actually the real deal. It then works well to strengthen muscles and build rigid, strong tissue. It might even assist with the loss of stubborn belly fat. Unfortunately, 1) the dosage needed makes it unimaginably toxic to the liver and 2) there is no actual medical use for it and cannot be taken for very long at all. For this reason, unless you are a bodybuilder willing to trade in your future health for accelerated, temporary gains now, Anavar should never be on your radar.

If you are looking for a supplement to take to maximize your masculine efficiency while ensuring a health-enjoying, long, quality life in the future, consider learning more about a bio-identical Testosterone replacement protocol.

Again, if you are considering bettering your health, changing your physique, improving your sex life and libido, consider a bio-identical testosterone replacement program. We can provide you the same type of testosterone your body produces. Of course, there is still the potential for side effects but, because it is bio-identical, your body is more receptive to it and you can continue therapy safely for many years. Most importantly, our doctors are experts in the art of Testosterone replacement therapy. We make sure our patients have all the required counterparts to their therapy to ensure better keeping unwanted side effects out of the picture.

A properly administered testosterone injection program can reignite the fire you have lost. It may sound intense, but the results of revitalizing the male body are unparalleled and near impossible to denote. It is simply a reawakening of life; a happier, more efficient life.

Testosterone Therapy Information

June 27, 2016 by admin 1 Comment

Testosterone Effects on Prostate 5 (1)

Testosterone Effects on Prostate Health.
How That’s Changing.

Testosterone effects on prostate levels and its functionality have always been a delicate topic of conversation. There is a lot of controversy surrounding the different theories on its impact. We have all heard the stories and read the articles where Testosterone injections, or, really any form of testosterone therapy, are allegedly attributed to malignant side effects in the participants. What is most interesting is the notion there actually have not been many studies done focused on that particular reaction to therapy. By this we mean, double-blind, placebo-controlled studies as is the standard of our trusted Food and Drug administration’s qualification process.

  • testosterone effects on prostateWe do not want to give off the impression we share this recently accepted hypothesis, which suggests testosterone effects on prostate health do not necessarily have a correlation. However, it is important to shed light on the fact the medical community and its core beliefs and socially accepted diagnoses often shift, and dramatically. Also, as the world awakens, begins to ask more questions and not accepting the typical status quo, we’ve all become a bit more enlightened to the ways of the world. We’ve learned that big pharma and its main directors don’t necessarily have our health or us at the forefront of their decision-making. Many times profits interfere with factual descriptions and publicly distributed medicinal allocations. We must use discernment when we read data and, dig deeper if it’s a matter of personal importance. Everyone who searches finds…

Particularly men on testosterone therapy, they should do their best research to help them better understand the involved variables. Better understanding of testosterone effects on prostate health can help patients make the best determination, based on their personal considering factors.

A recent study led by researchers at NYU Langone Medical Center denoted men prescribed a minimum of 1 year of testosterone injections replacement therapy demonstrated no additional risk for prostate cancer. One researcher, in particular, Stacy Loeb,testosterone effects on prostate MD, MSc, who happens to be a urologist at NYU Langone Medical Center shared a different notion. She suggests physicians should continue to evaluate prostate cancer risk factors and better understand functionality and future health— such as age over 40, family history of the disease and black ancestry — among men prescribed testosterone therapy. Dr. Loeb said in a corresponding press release, “they should not hesitate to prescribe [testosterone therapy] to appropriate patients for fear of increasing prostate cancer risk.”

In their research to better understand testosterone injections effects on prostate health, Dr. Loeb and her contemporaries collected data from close to 25 million medical files on a group of men. These men were enumerated in the National Prostate Cancer Register and Prescribed Drug Register in Sweden.

  • testosterone effects on prostateThey were able to classify 38,570 men diagnosed with prostate cancer between 2009 and 2012. Of these men, 284 received testosterone therapy treatments prior to their prostate tissue diagnosis. These numbers were then compared to the results of 192,838 men who showed no development of cancer in the prostate. Most interestingly, 1,378 of those men had been on testosterone therapy.
  • Multivariable examination helped conclude no significant association between any kind of testosterone injections replacement therapy and prostate cancer risk (OR = 1.03; 95% CI, 0.9-1.17). In fact, the results suggested a 35% increase in low-risk prostate cancer occurrence after the commencement of a testosterone therapy regimen (OR = 1.35; 95% CI, 1.16-1.56). Results also showed a lower risk of aggressive disease among men who used testosterone injections replacement therapy for more than a year (OR = 0.5; 95% CI, 0.37-0.67).

testosterone effects on prostateFrom this analysis, the demarcations indicating the lessened potential for testosterone injections therapy to have a negative side effect on the prostate are significantly prominent. Testosterone injections effects on prostate health and blood testing levels shown to have little to no association. That being said, until the concrete information has changed the national standard on this widespread opinion, AAI Rejuvenation Clinic will continue to supplement each one of our therapies with the necessary medicinal requirements currently utilized to maintain possible, unwanted side effects from interfering with a successful testosterone therapy treatment. If you have any questions or comments related to this subject, please fill out our short contact form and someone will reach you within 24 hours. Make sure to leave your phone number. You can also call us directly by dialing:

Testosterone Therapy Information

March 16, 2016 by admin 0 Comments

Testosterone Increasing Foods | Pineapple Testosterone Boosting Smoothie 5 (2)

Testosterone increasing Natural Options
(Testosterone Boosting Smoothies Series)

https://www.youtube.com/watch?v=ooyjsVpt_7E

Testosterone increasing smoothies are delicious and efficient. Today in our Testosterone Boosting Smoothies series, we want to incorporate a delicious contender. The all-mighty testosterone increasing Pineapple smoothie.

Before we begin, we want to thank all the readers for liking our postings and really taking a look at understanding the foundation of AAI Rejuvenation Clinic’s true intentions: To help everyone we touch better themselves in one way or another.

Of course, the nature of our business, our bread and butter are helping people balance their hormones correctly and Testosterone increasingeffectively through the efforts of bio-identical hormone programs like Testosterone therapyHGH therapy, and Sermorelin Therapy. However, before anyone jumps right into contemplating pills, supplements, nutraceuticals and pharmaceuticals, it’s important for this person with declining hormone levels to first aid the body in maximizing its own hormone production efforts.

Boosting your own Testosterone naturally before jumping right into an exogenous testosterone therapy will:

  • help your own endocrine system stay on track longer.
  • ensure other areas of your endocrine system aren’t prematurely hindered by the introduction of an external agent.
  • keep your whole body in better health, as the requirements would inevitably provide the results.
  • save hundreds if not thousands of dollars on therapy that could/should have been avoided.
  • ensure better overall results, as long as you are able to maintain “healthy” levels.

Remember that we are organic creatures. We like to mention this often in our posts. Absolutely: Western medicine has revolutionized humanity and extended our lives by more than double the original life expectancy. That being said, it’s unequivocal that it is also wreaking havoc on our biology. As many lives as we are able to save with western medication, there are equally as high and concerning numbers that can be drawn by counting victims of western medicine side effects. The point is, we need western medicine. We must have it, and in some cases, it is actually one of the real only hopes (such as blood pressure medication) however if we find natural, effective, organic options that can really help our health without the potentiality of hindering our internal systems, we have to take a look at them. Now, IMAGINE IF THEY TASTE GOOD TOO!!!

Testosterone increasing

There are real testosterone injections increasing options out there. Natural ones that are effective and cost-effective. Now, of course, there are situations where these options may not be sufficiently effective at maintaining your hormones at the level that they need to be to really maintain your health as you’ve always enjoyed it. In these cases, it’s important to try to qualify for therapy.

If you’d like to contact us to discuss today’s blog, other testosterone increasing options, or if you are interested in talking about the requirements for qualifying for hormone therapy, call us at:

 

Testosterone Therapy Information